Clinicopathological correlates of activating GNAS mutations in intraductal papillary mucinous neoplasm (IPMN) of the pancreas.

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Published in Ann Surg Oncol on July 12, 2013

Authors

Marco Dal Molin1,2, Hanno Matthaei1,3, Jian Wu4,5, Amanda Blackford6, Marija Debeljak1, Neda Rezaee6, Christopher L Wolfgang6,7, Giovanni Butturini2, Roberto Salvia2, Claudio Bassi2, Michael G Goggins1,7, Kenneth W Kinzler4,5, Bert Vogelstein4,5, James R Eshleman1,7, Ralph H Hruban1,7,6, Anirban Maitra1,7

Author Affiliations

1: Department of Pathology, The Sol Goldman Pancreatic Cancer Research Center, Johns Hopkins University School of Medicine, Baltimore, MD.
2: Unit of General Surgery B, Pancreas Institute, Department of Surgery, "G.B. Rossi" Hospital, University of Verona Hospital Trust, Verona, Italy.
3: Department of General, Visceral, Thoracic and Vascular Surgery, University of Bonn, Bonn, Germany.
4: Ludwig Center for Cancer Genetics, Johns Hopkins University School of Medicine, Baltimore, MD.
5: Howard Hughes Medical Institute, Johns Hopkins University School of Medicine, Baltimore, MD.
6: Department of Surgery, The Sol Goldman Pancreatic Cancer Research Center, Johns Hopkins University School of Medicine, Baltimore, MD.
7: Department of Oncology, The Sol Goldman Pancreatic Cancer Research Center, Johns Hopkins University School of Medicine, Baltimore, MD.

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