Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction.

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Published in J Am Heart Assoc on October 08, 2013

Authors

Marie-Hélène Disatnik1, Julio C B Ferreira, Juliane Cruz Campos, Kátia Sampaio Gomes, Paulo M M Dourado, Xin Qi, Daria Mochly-Rosen

Author Affiliations

1: Department of Chemical and Systems Biology, Stanford University School of Medicine, Stanford, 94305, CA.

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