PubRank

Increased prevalence of two mitochondrial DNA polymorphisms in functional disease: Are we describing different parts of an energy-depleted elephant?

PubWeight™: 0.81‹?›

🔗 View Article (PMID 25934187)

Published in Mitochondrion on April 29, 2015

Authors

Richard G Boles1, Essam A Zaki2, Jonathan R Kerr3, Kingshuk Das2, Sawona Biswas2, Ann Gardner4

Author Affiliations

1: Division of Medical Genetics and the Saban Research Institute, Children's Hospital Los Angeles, CA USA; Department of Pediatrics, Keck School of Medicine at the University of Southern California, Los Angeles, CA USA. Electronic address: richard.boles@courtagen.com.
2: Division of Medical Genetics and the Saban Research Institute, Children's Hospital Los Angeles, CA USA.
3: Division of Clinical Sciences, St. George's University of London, Cranmer Terrace, London United Kingdom.
4: Department of Clinical Neuroscience, Division of Psychiatry, Karolinska Institutet, Stockholm Sweden.

Associated clinical trials:

Sequenced Treatment Alternatives to Relieve Depression (STAR*D) | NCT00021528

Articles citing this ↘

Mitochondrial DNA variants correlate with symptoms in myalgic encephalomyelitis/chronic fatigue syndrome. J Transl Med (2016) 1.01

Clinically proven mtDNA mutations are not common in those with chronic fatigue syndrome. BMC Med Genet (2017) 0.75

The European ME/CFS Biomarker Landscape project: an initiative of the European network EUROMENE. J Transl Med (2017) 0.75