Angiogenin Mediates Cell-Autonomous Translational Control under Endoplasmic Reticulum Stress and Attenuates Kidney Injury.

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Published in J Am Soc Nephrol on July 20, 2015

Authors

Iadh Mami1, Nicolas Bouvier2, Khalil El Karoui3, Morgan Gallazzini3, Marion Rabant4, Pierre Laurent-Puig5, Shuping Li6, Pierre-Louis Tharaux7, Philippe Beaune5, Eric Thervet8, Eric Chevet9, Guo-Fu Hu6, Nicolas Pallet10

Author Affiliations

1: Institut National de la Sante et de la Recherche Médicale (INSERM) U1147, Saints-Pères Research Center Paris, France; Paris Descartes University Paris, France;
2: Nephrology Department Caen University Hospital, France;
3: Paris Descartes University Paris, France; INSERM U1151, Sick Childrens Necker Institute Paris, France;
4: Paris Descartes University Paris, France; Pathology Department, Necker Hospital Paris, France;
5: Institut National de la Sante et de la Recherche Médicale (INSERM) U1147, Saints-Pères Research Center Paris, France; Paris Descartes University Paris, France; Clinical Chemistry and.
6: Molecular Oncology Research Institute, Tufts Medical Center, Boston, Massachusetts;
7: Paris Cardiovascular Research Centre, INSERM, Paris, France; and.
8: Paris Descartes University Paris, France; Nephrology Departments, Georges Pompidou European Hospital Paris, France;
9: INSERM, UMR-U1053, Team Endoplasmic Reticulum Stress and Cancer, Bordeaux, France.
10: Institut National de la Sante et de la Recherche Médicale (INSERM) U1147, Saints-Pères Research Center Paris, France; Paris Descartes University Paris, France; Clinical Chemistry and Nephrology Departments, Georges Pompidou European Hospital Paris, France; npallet@yahoo.fr.

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