PubRank

PI3-Kinase-γ Has a Distinct and Essential Role in Lung-Specific Dendritic Cell Development.

PubWeight™: 0.78‹?›

🔗 View Article (PMID 26453378)

Published in Immunity on October 06, 2015

Authors

Samuel Philip Nobs1, Christoph Schneider1, Maren Gil Dietrich2, Thomas Brocker3, Antonius Rolink4, Emilio Hirsch5, Manfred Kopf6

Author Affiliations

1: Institute of Molecular Health Sciences, Department of Biology, ETH Zurich, 8093 Zurich, Switzerland.
2: Department of Endocrinology, Diabetes and Clinical Nutrition, University Hospital, 8091 Zurich, Switzerland.
3: Institute for Immunology, Ludwig-Maximillians-University Munich, 80336 Munich, Germany.
4: Department of Biomedicine, University Basel, 4058 Basel, Switzerland.
5: Department of Biomedicine, University Basel, 4058 Basel, Switzerland; Department of Molecular Biotechnology and Health Sciences, University of Torino, Via Nizza 52, 10126 Torino, Italy.
6: Institute of Molecular Health Sciences, Department of Biology, ETH Zurich, 8093 Zurich, Switzerland. Electronic address: manfred.kopf@ethz.ch.

Articles citing this ↘

Origin, Localization, and Immunoregulatory Properties of Pulmonary Phagocytes in Allergic Asthma. Front Immunol (2016) 0.82

PI3Kγ Is Critical for Dendritic Cell-Mediated CD8+ T Cell Priming and Viral Clearance during Influenza Virus Infection. PLoS Pathog (2016) 0.75

The Multifaceted Roles of PI3Kγ in Hypertension, Vascular Biology, and Inflammation. Int J Mol Sci (2016) 0.75

PPARγ in dendritic cells and T cells drives pathogenic type-2 effector responses in lung inflammation. J Exp Med (2017) 0.75

Articles by these authors ⇆

tEMPting Fate MaYBe the Solution. Immunity (2015) 0.75