Published in Acta Neuropathol on January 01, 1974
Significance of muscle biopsies in neuronal ceroid-lipofuscinoses. J Neurol Neurosurg Psychiatry (1975) 1.07
Effects of several lipidosis-including drugs upon the area postrema and adjacent medullary nuclei of adult rats. I. Alterations is perikarya and dendrites. Acta Neuropathol (1980) 0.86
Effects of several lipidosis-inducing drugs upon the area postrema and adjacent medullary nuclei of adult rats. II. Axonal alternations. Acta Neuropathol (1981) 0.84
Effects of chloroquine on neurons of long-term cultures of peripheral and central nervous system. A light and electron microscope study. Acta Neuropathol (1974) 0.79
Differential effects of chloroquine and of several other amphiphilic cationic drugs upon rat choroid plexus. Acta Neuropathol (1979) 0.78
[Tumor- and drug-induced cutaneous axonal dystrophy. An electronmicrocopy proof of multiple lamellated bodies]. Arch Dermatol Res (1975) 0.75
Uptake of tritium labelled chloroquine into organized cultures of rat spinal ganglia. An electron microscope autoradiographic study. Acta Neuropathol (1975) 0.75
Changes caused by chloroquine in cultured nervous tissues of the rat. Acta Neuropathol (1975) 0.75
A SIMPLIFIED LEAD CITRATE STAIN FOR USE IN ELECTRON MICROSCOPY. J Cell Biol (1965) 37.37
CHROMATOGRAPHIC SEPARATION OF HUMAN BRAIN GANGLIOSIDES. J Neurochem (1963) 6.22
UPTAKE OF DYES AND DRUGS BY LIVING CELLS IN CULTURE. Life Sci (1964) 4.54
CHLOROQUINE NEUROMYOPATHY. Proc Staff Meet Mayo Clin (1963) 2.84
LABILIZATION AND STABILIZATION OF LYSOSOMES. Fed Proc (1965) 2.05
Lysosomes and joint disease. Arthritis Rheum (1966) 1.90
Neuropathological conditions related to neuroaxonal dystrophy. Acta Neuropathol (1971) 1.75
Acid maltase deficiency in adults: studies in four cases of a syndrome which may mimic muscular dystrophy or other myopathies. Brain (1970) 1.72
Studies on vascular permeability in peripheral nerves. IV. Distribution of intravenously injected protein tracers in the peripheral nervous system of various species. Acta Neuropathol (1971) 1.68
Chloroquine myopathy. Q J Med (1971) 1.56
THE OCULAR DEPOSITION OF CHLOROQUINE. Invest Ophthalmol (1963) 1.56
Chloroquine: mode of action. Science (1966) 1.55
Effect of acetylsalicylic acid on lysosomes. Proc Soc Exp Biol Med (1966) 1.52
Experimental chloroquine myopathy. J Neurol Neurosurg Psychiatry (1966) 1.50
Experimental study of diphtheritic polyneuritis in the rabbit and guinea pig. III. The bloodnerve barrier in the rabbit. J Neuropathol Exp Neurol (1961) 1.48
CHLOROQUINE NEUROMYOPATHY. Br Med J (1964) 1.33
Nervous system in Pompe's disease. Ultrastructure and biochemistry. J Neuropathol Exp Neurol (1971) 1.31
Experimental chloroquine myopathy. J Neuropathol Exp Neurol (1970) 1.30
VACUOLAR MYOPATHY: CLINICAL, HISTOCHEMICAL, AND MICROSCOPIC STUDY. Arch Neurol (1965) 1.28
Pompe's disease: an inborn lysosomal disorder with storage of glycogen. A study of brain and striated muscle. Acta Neuropathol (1973) 1.24
PERIPHERAL NEUROPATHY FOLLOWING CHLOROQUINE THERAPY. Can Med Assoc J (1963) 1.23
Lysosomes. N Engl J Med (1965) 1.19
Ultrastructure of the central nervous system after prolonged hypoxia. I. Neuronal alterations. Acta Neuropathol (1972) 1.19
Formation of myeloid bodies in rat liver lysosomes after chloroquine administration. Exp Mol Pathol (1968) 1.11
Study of chloroquine toxicity and a drug-induced cerebrospinal lipodystrophy in swine. Am J Pathol (1968) 1.10
Basic cellular lesion in chloroquine toxicity. Lab Invest (1971) 1.09
Irreversible lysosomal damage induced by chloroquine in the retinae of pigmented and albino rats. Exp Mol Pathol (1970) 1.05
Lipofuscin pigment accumulation as a function of age and distribution in rodent brain. J Gerontol (1968) 1.02
Accumulation of a glycerolphospholipid in classical niemann-pick disease. Lipids (1968) 1.02
Chloroquine myopathy. J Neurol Neurosurg Psychiatry (1966) 1.01
Chloroquine-induced alterations in rat sensory ganglia cultivated in vitro. A light and electron microscope study. Acta Neuropathol (1972) 0.99
Effects of chronic chloroquine treatment on lysosomes of rat liver cells. Exp Mol Pathol (1970) 0.98
Fine structure and chemical composition of opaque cytoplasmic bodies of triparanol treated Syrian hamsters. II. Phospholipid analysis of opaque bodies from adrenal glands. Tex Rep Biol Med (1967) 0.98
[Drug-induced phospholipid storage disease]. Dtsch Med Wochenschr (1973) 0.95
Chloroquine neuromyopathy, a reversible granulovascuolar myopathy. Arch Pathol (1972) 0.93
Drug-induced lipidosis in human cases and in animal experiments. Accumulation of an acidic glycerophospholipid. J Biochem (1971) 0.89
The induction of ganglioside storage in nervous system cultures. Lab Invest (1972) 0.89
The effect of chloroquine on rat heart lysosomes. J Ultrastruct Res (1969) 0.88
Chloroquine neuromyopathy. Ann Phys Med (1968) 0.88
FINE STRUCTURAL LOCALIZATION OF ACID PHOSPHATASE ACTIVITY IN NEURONS OF TAY-SACHS DISEASE. J Neuropathol Exp Neurol (1964) 0.86
[On the feeding of laboratory animals. 1. Altraomin R--a standard diet for rats and mice]. Arzneimittelforschung (1961) 0.84
Accumulation of acidic phospholipids in a case of hyperlipidemia with hepatosplenomegaly. Lipids (1970) 0.81
[Histopathology of polyneuritis and polyneuropathy]. Dtsch Z Nervenheilkd (1959) 0.81
Chloroquine-induced myopathy. Br J Dermatol (1969) 0.80
[Infantile amaurotic idiocy and related forms as ganglioside storage diseases]. Naturwissenschaften (1969) 0.79
Tissue distribution of chloroquine in rats in the course of long-term application. Arch Int Pharmacodyn Ther (1972) 0.79
[Autoradiographic study of the rate of protein synthesis in red and white muscle fibers]. Exp Cell Res (1967) 0.79
Peripheral neuromyopathy following chloroquine therapy. Med J Aust (1966) 0.79
[Experimental studies on the etiology of nitrofurane-induced polyneuropathy and histopathology of chlorochindiphosphate (Resochin) neuro-myopathy]. Beitr Neurochir (1970) 0.79
[Tay-Sachs' amaurotic idiocy]. Presse Med (1953) 0.77
[Histopathologic peculiarities of the myoneuropathy caused by chloroquine diphosphate]. Naturwissenschaften (1968) 0.77
[Electromyography and chloroquine-induced neuromyopathy]. Rev Neurol (Paris) (1968) 0.77
REVERSIBLE MYOPATHY IN CHLOROQUINE TREATMENT. Acta Med Scand (1965) 0.77
[Cerebral sphingolipidoses as inborn errors of metabolism]. Dtsch Med Wochenschr (1970) 0.76
Chloroquine-induced accumulation of gangliosides and phospholipids in skeletal muscles. Quantitative determination and characterization of stored lipids. Eur J Biochem (1981) 0.85
Effect of chloroquine on the activity of some lysosomal enzymes involved in ganglioside degradation. Biochim Biophys Acta (1987) 0.83
[Experimental studies on the etiology of nitrofurane-induced polyneuropathy and histopathology of chlorochindiphosphate (Resochin) neuro-myopathy]. Beitr Neurochir (1970) 0.79
[Lysosomes in experimental storage dystrophy due to chronic chloroquine intoxication (author's transl)]. Verh Dtsch Ges Pathol (1976) 0.78
[Damage to the nervous system due to nitrofurans in the rat]. Acta Neuropathol (1967) 0.78
Chloroquine intoxication induces ganglioside storage in nervous tissue: a chemical and histopathological study of brain, spinal cord, dorsal root ganglia, and retinal in the miniature pig. J Neurochem (1981) 0.77
[Histopathologic peculiarities of the myoneuropathy caused by chloroquine diphosphate]. Naturwissenschaften (1968) 0.77
Lipid accumulation in liver, spleen, lungs and kidneys of miniature-pigs after chloroquine treatment. Biochem J (1982) 0.76
Experimental neuropathology and clinical neurology of chloroquine side effects. Int J Neurol (1978) 0.75
[Wilhelm Krücke (1911-1988): on the occasion of the 100th birthday of the great neuropathologist on December 26, 2011]. Fortschr Neurol Psychiatr (2011) 0.75
[Pathology of neurons -- preferences and differences in the neuronal attack system in experimental radial nerve dystrophy induced by chloroquine]. Acta Histochem Suppl (1981) 0.75
Chemical structures of three fucogangliosides isolated from nervous tissue of mini-pig. Eur J Biochem (1981) 0.75
[Relations between polyneuropathies produced by certain modern drugs, by malabsorption and by malnutrition]. Verh Dtsch Ges Pathol (1969) 0.75