| Rank | Title | Journal | Year | PubWeight™‹?› |
|---|---|---|---|---|
| 1 | The majority of epidermal T cells in Psoriasis vulgaris lesions can produce type 1 cytokines, interferon-gamma, interleukin-2, and tumor necrosis factor-alpha, defining TC1 (cytotoxic T lymphocyte) and TH1 effector populations: a type 1 differentiation bias is also measured in circulating blood T cells in psoriatic patients. | J Invest Dermatol | 1999 | 2.31 |
| 2 | Molecular classification of psoriasis disease-associated genes through pharmacogenomic expression profiling. | Pharmacogenomics J | 2001 | 1.34 |
| 3 | Interleukin-11 therapy selectively downregulates type I cytokine proinflammatory pathways in psoriasis lesions. | J Clin Invest | 1999 | 1.21 |
| 4 | Successful in vivo blockade of CD25 (high-affinity interleukin 2 receptor) on T cells by administration of humanized anti-Tac antibody to patients with psoriasis. | J Am Acad Dermatol | 2000 | 1.07 |
| 5 | PUVA-induced lymphocyte apoptosis: mechanism of action in psoriasis. | Photodermatol Photoimmunol Photomed | 1999 | 0.80 |