Published in J Mol Cell Cardiol on November 01, 1999
Regulation of intracellular and mitochondrial sodium in health and disease. Circ Res (2009) 1.24
The cardiac persistent sodium current: an appealing therapeutic target? Br J Pharmacol (2007) 1.23
Ionic mechanisms of cardiac cell swelling induced by blocking Na+/K+ pump as revealed by experiments and simulation. J Gen Physiol (2006) 0.98
Ion transport and energetics during cell death and protection. Physiology (Bethesda) (2008) 0.95
Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial function. J Cardiovasc Pharmacol (2008) 0.94
Riluzole protects against cardiac ischaemia and reperfusion damage via block of the persistent sodium current. Br J Pharmacol (2010) 0.90
Triple threat: the Na+/Ca2+ exchanger in the pathophysiology of cardiac arrhythmia, ischemia and heart failure. Curr Drug Targets (2011) 0.90
Why did the NHE inhibitor clinical trials fail? J Mol Cell Cardiol (2008) 0.90
Persistent sodium current and Na+/H+ exchange contributes to the augmentation of the reverse Na+/Ca2+ exchange during hypoxia or acute ischemia in ventricular myocytes. Pflugers Arch (2012) 0.90
The rise of [Na(+)] (i) during ischemia and reperfusion in the rat heart-underlying mechanisms. Pflugers Arch (2007) 0.89
Preservation of mitochondrial function may contribute to cardioprotective effects of Na+/Ca2+ exchanger inhibitors in ischaemic/reperfused rat hearts. Br J Pharmacol (2007) 0.82
Temperature dependence of Na+-H+ exchange, Na+-HCO3- co-transport, intracellular buffering and intracellular pH in guinea-pig ventricular myocytes. J Physiol (2003) 0.81
A model of Na+/H+ exchanger and its central role in regulation of pH and Na+ in cardiac myocytes. Biophys J (2009) 0.80
Na/H exchange inhibition protects newborn heart from ischemia/reperfusion injury by limiting Na+-dependent Ca2+ overload. J Cardiovasc Pharmacol (2010) 0.80
Cariporide enhances lactate clearance upon reperfusion but does not alter lactate accumulation during global ischaemia. Pflugers Arch (2003) 0.78
Regulation of ion gradients across myocardial ischemic border zones: a biophysical modelling analysis. PLoS One (2013) 0.78
R 56865 exerts cardioprotective properties independent of the intracellular Na(+)-overload in the guinea pig heart. Naunyn Schmiedebergs Arch Pharmacol (2003) 0.76
Na(+)/H(+) exchanger in the regulation of platelet activation and paradoxical effects of cariporide. Exp Neurol (2015) 0.75
Blowing off acid: a new tool to study Na+/HCO3- co-transport. Br J Pharmacol (2008) 0.75
Myoglobin: A scavenger of bioactive NO. Proc Natl Acad Sci U S A (2001) 2.26
Coronary hemodynamics in endothelial NO synthase knockout mice. Circ Res (1998) 1.80
Disruption of myoglobin in mice induces multiple compensatory mechanisms. Proc Natl Acad Sci U S A (1999) 1.60
Inhibition of adenosine kinase increases endogenous adenosine and depresses neuronal activity in hippocampal slices. Neuropharmacology (1994) 1.15
Contribution of NO to ischemia-reperfusion injury in the saline-perfused heart: a study in endothelial NO synthase knockout mice. J Mol Cell Cardiol (1999) 1.10
Myoglobin facilitates oxygen diffusion. FASEB J (2001) 1.05
Inotropic response to beta-adrenergic receptor stimulation and anti-adrenergic effect of ACh in endothelial NO synthase-deficient mouse hearts. J Physiol (2001) 0.99
Rapid turnover of the AMP-adenosine metabolic cycle in the guinea pig heart. Circ Res (1993) 0.86
Cloning of the rat P2u receptor and its potential role in coronary vasodilation. Am J Physiol (1996) 0.85
Nitric oxide induced contractile dysfunction is related to a reduction in myocardial energy generation. Cardiovasc Res (1997) 0.84
Metabolic adaptation of endothelial cells to substrate deprivation. Am J Physiol (1999) 0.79
Nitric oxide reduces energy supply by direct action on the respiratory chain in isolated cardiomyocytes. Am J Physiol Heart Circ Physiol (2001) 0.79
Decrease of ischemic injury to the isolated perfused rat liver by loop diuretics. Hepatology (1997) 0.77
Cardioprotective actions of KC 12291. II. Delaying Na+ overload in ischemia improves cardiac function and energy status in reperfusion. Naunyn Schmiedebergs Arch Pharmacol (1998) 0.76
Purinogen is not an endogenous substrate used in endothelial cells during substrate deprivation. Biochem J (1999) 0.75