Adaptive mutations in the V3 loop of gp120 enhance fusogenicity of human immunodeficiency virus type 1 and enable use of a CCR5 coreceptor that lacks the amino-terminal sulfated region.

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Published in J Virol on December 01, 2001

Authors

E J Platt1, S E Kuhmann, P P Rose, D Kabat

Author Affiliations

1: Department of Biochemistry and Molecular Biology, Oregon Health Sciences University, Portland, Oregon 97201-3098, USA.

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