PubRank

Horacio E Cingolani

Author PubWeight™ 26.34‹?›

Top papers

Rank Title Journal Year PubWeight™‹?›
1 Endurance training in the spontaneously hypertensive rat: conversion of pathological into physiological cardiac hypertrophy. Hypertension 2009 1.22
2 Na+/H+ exchanger-1 inhibitors decrease myocardial superoxide production via direct mitochondrial action. J Appl Physiol (1985) 2008 0.98
3 Myocardial hypertrophy of normotensive Wistar-Kyoto rats. Am J Physiol Heart Circ Physiol 2003 0.97
4 Phosphodiesterase 5A inhibition induces Na+/H+ exchanger blockade and protection against myocardial infarction. Hypertension 2007 0.92
5 Mitochondrial reactive oxygen species activate the slow force response to stretch in feline myocardium. J Physiol 2007 0.91
6 Aldosterone stimulates the cardiac Na(+)/H(+) exchanger via transactivation of the epidermal growth factor receptor. Hypertension 2011 0.91
7 The Anrep effect requires transactivation of the epidermal growth factor receptor. J Physiol 2010 0.91
8 Regression of isoproterenol-induced cardiac hypertrophy by Na+/H+ exchanger inhibition. Hypertension 2003 0.90
9 Silencing of cardiac mitochondrial NHE1 prevents mitochondrial permeability transition pore opening. Am J Physiol Heart Circ Physiol 2011 0.88
10 Regression of cardiomyocyte hypertrophy in SHR following chronic inhibition of the Na(+)/H(+) exchanger. Cardiovasc Res 2002 0.88
11 Normalization of the calcineurin pathway underlies the regression of hypertensive hypertrophy induced by Na+/H+ exchanger-1 (NHE-1) inhibition. Can J Physiol Pharmacol 2007 0.87
12 Endothelin isoforms and the response to myocardial stretch. Am J Physiol Heart Circ Physiol 2005 0.86
13 Effects of different fractions of a red wine non-alcoholic extract on ischemia-reperfusion injury. Life Sci 2005 0.85
14 Cardioprotection from ischemia/reperfusion induced by red wine extract is mediated by K(ATP) channels. J Cardiovasc Pharmacol 2002 0.85
15 Na+/H+ exchanger inhibition at the onset of reperfusion decreases myocardial infarct size: role of reactive oxygen species. Cardiovasc Pathol 2006 0.85
16 A low dose of angiotensin II increases inotropism through activation of reverse Na(+)/Ca(2+) exchange by endothelin release. Cardiovasc Res 2003 0.85
17 Myocardial reperfusion injury: reactive oxygen species vs. NHE-1 reactivation. Cell Physiol Biochem 2011 0.82
18 In vivo key role of reactive oxygen species and NHE-1 activation in determining excessive cardiac hypertrophy. Pflugers Arch 2011 0.82
19 Autocrine stimulation of cardiac Na(+)-Ca(2+) exchanger currents by endogenous endothelin released by angiotensin II. Circ Res 2002 0.82
20 Silencing of NHE-1 blunts the slow force response to myocardial stretch. J Appl Physiol (1985) 2011 0.81
21 Mineralocorticoid receptor activation is crucial in the signalling pathway leading to the Anrep effect. J Physiol 2011 0.81
22 Endogenous endothelin 1 mediates angiotensin II-induced hypertrophy in electrically paced cardiac myocytes through EGFR transactivation, reactive oxygen species and NHE-1. Pflugers Arch 2013 0.80
23 [Angiotensin II-induced endothelin-1 release in cardiac myocytes]. Medicina (B Aires) 2006 0.79
24 Na(+)-H(+) exchanger inhibition: a new antihypertrophic tool. Circ Res 2002 0.79
25 Upregulation of myocardial Na+/H+ exchanger induced by chronic treatment with a selective inhibitor. J Mol Cell Cardiol 2002 0.77
26 Silencing of sodium/hydrogen exchanger in the heart by direct injection of naked siRNA. J Appl Physiol (1985) 2011 0.77
27 The autocrine/paracrine loop after myocardial stretch: mineralocorticoid receptor activation. Curr Cardiol Rev 2013 0.77
28 Myocardial mineralocorticoid receptor activation by stretching and its functional consequences. Hypertension 2013 0.77
29 A possible subcellular mechanism underlying the "French paradox": the opening of mitochondrial K(ATP) channels. Appl Physiol Nutr Metab 2011 0.77
30 Phosphodiesterase 5A inhibition decreases NHE-1 activity without altering steady state pH(i): role of phosphatases. Cell Physiol Biochem 2010 0.76
31 Letter by Cingolani et al regarding article, "Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice". Circulation 2009 0.75
32 Comparison of the effects of different magnesium administration times on infarct size. Exp Clin Cardiol 2003 0.75
33 Endothelin 1 versus endothelin 3 in the development of the slow force response to myocardial stretch. Can J Cardiol 2005 0.75
34 Effects of antihypertensive therapy on cardiac sodium/hydrogen ion exchanger activity and hypertrophy in spontaneously hypertensive rats. Can J Cardiol 2002 0.75
35 [Basic research in Medicine]. Medicina (B Aires) 2010 0.75
36 Decreased activity of the Na+/H+ exchanger by phosphodiesterase 5A inhibition is attributed to an increase in protein phosphatase activity. Hypertension 2010 0.75