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Genome sequence of Streptococcus agalactiae, a pathogen causing invasive neonatal disease.
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2004
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Type II fatty acid synthesis is not a suitable antibiotic target for Gram-positive pathogens.
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Sorting sortases: a nomenclature proposal for the various sortases of Gram-positive bacteria.
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Res Microbiol
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Dual role for pilus in adherence to epithelial cells and biofilm formation in Streptococcus agalactiae.
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PLoS Pathog
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Formation of D-alanyl-lipoteichoic acid is required for adhesion and virulence of Listeria monocytogenes.
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Mol Microbiol
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2002
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Shaping a bacterial genome by large chromosomal replacements, the evolutionary history of Streptococcus agalactiae.
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Taxonomic dissection of the Streptococcus bovis group by analysis of manganese-dependent superoxide dismutase gene (sodA) sequences: reclassification of 'Streptococcus infantarius subsp. coli' as Streptococcus lutetiensis sp. nov. and of Streptococcus bovis biotype 11.2 as Streptococcus pasteurianus sp. nov.
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2002
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TLR-independent type I interferon induction in response to an extracellular bacterial pathogen via intracellular recognition of its DNA.
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2008
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11
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CovS/CovR of group B streptococcus: a two-component global regulatory system involved in virulence.
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Multiplex PCR assay for rapid and accurate capsular typing of group B streptococci.
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Microbes Infect
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The SrtA Sortase of Streptococcus agalactiae is required for cell wall anchoring of proteins containing the LPXTG motif, for adhesion to epithelial cells, and for colonization of the mouse intestine.
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Infect Immun
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2005
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Genome sequence of Streptococcus gallolyticus: insights into its adaptation to the bovine rumen and its ability to cause endocarditis.
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J Bacteriol
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Attenuated virulence of Streptococcus agalactiae deficient in D-alanyl-lipoteichoic acid is due to an increased susceptibility to defensins and phagocytic cells.
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Mol Microbiol
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2003
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17
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The surface protein HvgA mediates group B streptococcus hypervirulence and meningeal tropism in neonates.
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J Exp Med
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1.51
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Lipoproteins are critical TLR2 activating toxins in group B streptococcal sepsis.
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J Immunol
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19
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Role of lipoteichoic acid in the phagocyte response to group B streptococcus.
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J Immunol
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Rapid detection of the "highly virulent" group B Streptococcus ST-17 clone.
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Microbes Infect
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Cell surface of Lactococcus lactis is covered by a protective polysaccharide pellicle.
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J Biol Chem
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22
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Molecular dissection of the secA2 locus of group B Streptococcus reveals that glycosylation of the Srr1 LPXTG protein is required for full virulence.
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J Bacteriol
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1.27
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Activation of the NLRP3 inflammasome by group B streptococci.
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J Immunol
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Respiration metabolism of Group B Streptococcus is activated by environmental haem and quinone and contributes to virulence.
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Mol Microbiol
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1.24
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Genetic basis of antibiotic resistance in Streptococcus agalactiae strains isolated in a French hospital.
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Antimicrob Agents Chemother
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The putative glycosyltransferase-encoding gene cylJ and the group B Streptococcus (GBS)-specific gene cylK modulate hemolysin production and virulence of GBS.
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Infect Immun
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2007
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1.22
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27
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Streptococcus agalactiae GAPDH is a virulence-associated immunomodulatory protein.
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J Immunol
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Invasive group B streptococcal infections in infants, France.
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Emerg Infect Dis
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Capsular switching in group B Streptococcus CC17 hypervirulent clone: a future challenge for polysaccharide vaccine development.
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J Infect Dis
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Native valve endocarditis due to Enterococcus hirae.
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The Group B Streptococcus NADH oxidase Nox-2 is involved in fatty acid biosynthesis during aerobic growth and contributes to virulence.
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Mol Microbiol
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1.07
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32
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D-alanylation of lipoteichoic acids confers resistance to cationic peptides in group B streptococcus by increasing the cell wall density.
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PLoS Pathog
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1.05
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Atypical association of DDE transposition with conjugation specifies a new family of mobile elements.
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Mol Microbiol
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2008
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Molecular characterization of a Streptococcus gallolyticus genomic island encoding a pilus involved in endocarditis.
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J Infect Dis
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Group B streptococcus GAPDH is released upon cell lysis, associates with bacterial surface, and induces apoptosis in murine macrophages.
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An in silico model for identification of small RNAs in whole bacterial genomes: characterization of antisense RNAs in pathogenic Escherichia coli and Streptococcus agalactiae strains.
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Rapid and accurate identification of human isolates of Pasteurella and related species by sequencing the sodA gene.
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The 2-Cys peroxiredoxin alkyl hydroperoxide reductase c binds heme and participates in its intracellular availability in Streptococcus agalactiae.
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Interaction with human plasminogen system turns on proteolytic activity in Streptococcus agalactiae and enhances its virulence in a mouse model.
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Role of the Group B antigen of Streptococcus agalactiae: a peptidoglycan-anchored polysaccharide involved in cell wall biogenesis.
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Genetic basis of antibiotic resistance in clinical isolates of Streptococcus gallolyticus (Streptococcus bovis).
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Fluoroquinolone-resistant group B streptococci in acute exacerbation of chronic bronchitis.
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Roles of environmental heme, and menaquinone, in streptococcus agalactiae.
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Biometals
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Inhibition of IL-10 production by maternal antibodies against Group B Streptococcus GAPDH confers immunity to offspring by favoring neutrophil recruitment.
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PLoS Pathog
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Group B Streptococcus surface proteins as major determinants for meningeal tropism.
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Curr Opin Microbiol
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The Abi-domain protein Abx1 interacts with the CovS histidine kinase to control virulence gene expression in group B Streptococcus.
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PLoS Pathog
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Adult zebrafish model of bacterial meningitis in Streptococcus agalactiae infection.
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Dev Comp Immunol
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Extracellular nucleotide catabolism by the Group B Streptococcus ectonucleotidase NudP increases bacterial survival in blood.
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Effect of PhoP-PhoQ activation by broad repertoire of antimicrobial peptides on bacterial resistance.
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Role of the Streptococcus agalactiae ClpP serine protease in heat-induced stress defence and growth arrest.
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The GBS PI-2a pilus is required for virulence in mice neonates.
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Epidemiologically and clinically relevant Group B Streptococcus isolates do not bind collagen but display enhanced binding to human fibrinogen.
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Molecular mapping of the cell wall polysaccharides of the human pathogen Streptococcus agalactiae.
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FbsC, a novel fibrinogen-binding protein, promotes Streptococcus agalactiae-host cell interactions.
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Group B Streptococcus hijacks the host plasminogen system to promote brain endothelial cell invasion.
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TLR2-induced IL-10 production impairs neutrophil recruitment to infected tissues during neonatal bacterial sepsis.
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J Immunol
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Rga, a RofA-like regulator, is the major transcriptional activator of the PI-2a pilus in Streptococcus agalactiae.
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Comparison of the Diversilab® system with multi-locus sequence typing and pulsed-field gel electrophoresis for the characterization of Streptococcus agalactiae invasive strains.
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SecA localization and SecA-dependent secretion occurs at new division septa in group B Streptococcus.
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Analysis of the Streptococcus agalactiae exoproteome.
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Comparative evaluation of VITEK 2 for antimicrobial susceptibility testing of group B Streptococcus.
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Molecular basis for different levels of tet(M) expression in Streptococcus pneumoniae clinical isolates.
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Construction of isogenic mutants in Streptococcus gallolyticus based on the development of new mobilizable vectors.
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