Peter J Minogue

Author PubWeight™ 14.61^‹?›

Top papers

Rank	Title	Journal	Year	PubWeight™‹?›
1	Loss of function and impaired degradation of a cataract-associated mutant connexin50.	Eur J Cell Biol	2003	1.48
2	Pathways for degradation of connexins and gap junctions.	Cardiovasc Res	2004	1.38
3	Autophagy: a pathway that contributes to connexin degradation.	J Cell Sci	2011	1.17
4	Different consequences of cataract-associated mutations at adjacent positions in the first extracellular boundary of connexin50.	Am J Physiol Cell Physiol	2011	1.02
5	Cataracts are caused by alterations of a critical N-terminal positive charge in connexin50.	Invest Ophthalmol Vis Sci	2008	1.01
6	An intact connexin N-terminus is required for function but not gap junction formation.	J Cell Sci	2008	0.95
7	An MIP/AQP0 mutation with impaired trafficking and function underlies an autosomal dominant congenital lamellar cataract.	Exp Eye Res	2012	0.93
8	Transgenic overexpression of connexin50 induces cataracts.	Exp Eye Res	2007	0.90
9	Cytoplasmic amino acids within the membrane interface region influence connexin oligomerization.	J Membr Biol	2012	0.89
10	Different domains are critical for oligomerization compatibility of different connexins.	Biochem J	2011	0.89
11	Connexin50D47A decreases levels of fiber cell connexins and impairs lens fiber cell differentiation.	Invest Ophthalmol Vis Sci	2013	0.89
12	The GJA8 allele encoding CX50I247M is a rare polymorphism, not a cataract-causing mutation.	Mol Vis	2009	0.87
13	The N terminus of connexin37 contains an alpha-helix that is required for channel function.	J Biol Chem	2009	0.87
14	Highly restricted pattern of connexin36 expression in chick somite development.	Anat Embryol (Berl)	2004	0.82
15	The connexin46 mutant, Cx46T19M, causes loss of gap junction function and alters hemi-channel gating.	J Membr Biol	2014	0.76