| Rank |
Title |
Journal |
Year |
PubWeight™‹?› |
|
1
|
Slc26a11, a chloride transporter, localizes with the vacuolar H(+)-ATPase of A-intercalated cells of the kidney.
|
Kidney Int
|
2011
|
2.21
|
|
2
|
slc26a3 (dra)-deficient mice display chloride-losing diarrhea, enhanced colonic proliferation, and distinct up-regulation of ion transporters in the colon.
|
J Biol Chem
|
2006
|
2.20
|
|
3
|
Renal and intestinal transport defects in Slc26a6-null mice.
|
Am J Physiol Cell Physiol
|
2004
|
1.68
|
|
4
|
Hypoxia regulates expression and activity of Kv1.3 channels in T lymphocytes: a possible role in T cell proliferation.
|
J Immunol
|
2003
|
1.31
|
|
5
|
Identification of thrombospondin 1 (TSP-1) as a novel mediator of cell injury in kidney ischemia.
|
J Clin Invest
|
2005
|
1.19
|
|
6
|
Deletion of the chloride transporter Slc26a9 causes loss of tubulovesicles in parietal cells and impairs acid secretion in the stomach.
|
Proc Natl Acad Sci U S A
|
2008
|
1.18
|
|
7
|
Double knockout of pendrin and Na-Cl cotransporter (NCC) causes severe salt wasting, volume depletion, and renal failure.
|
Proc Natl Acad Sci U S A
|
2012
|
1.16
|
|
8
|
SLC26A9 is expressed in gastric surface epithelial cells, mediates Cl-/HCO3- exchange, and is inhibited by NH4+.
|
Am J Physiol Cell Physiol
|
2005
|
1.15
|
|
9
|
Sodium and chloride absorptive defects in the small intestine in Slc26a6 null mice.
|
Pflugers Arch
|
2007
|
1.04
|
|
10
|
Deletion of the anion exchanger Slc26a4 (pendrin) decreases apical Cl(-)/HCO3(-) exchanger activity and impairs bicarbonate secretion in kidney collecting duct.
|
Am J Physiol Cell Physiol
|
2010
|
1.02
|
|
11
|
SLC26A7: a basolateral Cl-/HCO3- exchanger specific to intercalated cells of the outer medullary collecting duct.
|
Am J Physiol Renal Physiol
|
2003
|
1.01
|
|
12
|
Identification of a basolateral Cl-/HCO3- exchanger specific to gastric parietal cells.
|
Am J Physiol Gastrointest Liver Physiol
|
2003
|
1.01
|
|
13
|
Identification of an apical Cl-/HCO3- exchanger in gastric surface mucous and duodenal villus cells.
|
Am J Physiol Gastrointest Liver Physiol
|
2003
|
1.00
|
|
14
|
Expression of SSAT, a novel biomarker of tubular cell damage, increases in kidney ischemia-reperfusion injury.
|
Am J Physiol Renal Physiol
|
2003
|
0.99
|
|
15
|
Stathmin-deficient mice develop fibrosis and show delayed recovery from ischemic-reperfusion injury.
|
Am J Physiol Renal Physiol
|
2006
|
0.99
|
|
16
|
Deletion of the chloride transporter slc26a7 causes distal renal tubular acidosis and impairs gastric acid secretion.
|
J Biol Chem
|
2009
|
0.95
|
|
17
|
Overexpression of SSAT in kidney cells recapitulates various phenotypic aspects of kidney ischemia-reperfusion injury.
|
J Am Soc Nephrol
|
2004
|
0.94
|
|
18
|
Proximal tubule specific knockout of the Na⁺/H⁺ exchanger NHE3: effects on bicarbonate absorption and ammonium excretion.
|
J Mol Med (Berl)
|
2013
|
0.93
|
|
19
|
Potentiation of the effect of thiazide derivatives by carbonic anhydrase inhibitors: molecular mechanisms and potential clinical implications.
|
PLoS One
|
2013
|
0.90
|
|
20
|
Spermidine/spermine-N1-acetyltransferase ablation protects against liver and kidney ischemia-reperfusion injury in mice.
|
Am J Physiol Gastrointest Liver Physiol
|
2009
|
0.90
|
|
21
|
Identification of stathmin as a novel marker of cell proliferation in the recovery phase of acute ischemic renal failure.
|
Am J Physiol Cell Physiol
|
2003
|
0.87
|
|
22
|
The role of spermidine/spermine N1-acetyltransferase in endotoxin-induced acute kidney injury.
|
Am J Physiol Cell Physiol
|
2010
|
0.86
|
|
23
|
Slc26a6 (PAT1) deletion downregulates the apical Na+/H+ exchanger in the straight segment of the proximal tubule.
|
Am J Nephrol
|
2007
|
0.82
|
|
24
|
The chloride channel/transporter Slc26a9 regulates the systemic arterial pressure and renal chloride excretion.
|
J Mol Med (Berl)
|
2012
|
0.81
|
|
25
|
Double knockout of carbonic anhydrase II (CAII) and Na(+)-Cl(-) cotransporter (NCC) causes salt wasting and volume depletion.
|
Cell Physiol Biochem
|
2013
|
0.78
|
|
26
|
Activation of the apical Na+/H+ exchanger NHE3 by formate: a basis of enhanced fluid and electrolyte reabsorption by formate in the kidney.
|
Am J Physiol Renal Physiol
|
2004
|
0.78
|
|
27
|
Slc26a11 is prominently expressed in the brain and functions as a chloride channel: expression in Purkinje cells and stimulation of V H⁺-ATPase.
|
Pflugers Arch
|
2013
|
0.78
|
|
28
|
Vasopressin induces expression of the Cl-/HCO3- exchanger SLC26A7 in kidney medullary collecting ducts of Brattleboro rats.
|
Am J Physiol Renal Physiol
|
2005
|
0.76
|