Published in Circulation on March 01, 1992
Rate dependence and regulation of action potential and calcium transient in a canine cardiac ventricular cell model. Circulation (2004) 2.83
Connexin43 gene transfer reduces ventricular tachycardia susceptibility after myocardial infarction. J Am Coll Cardiol (2012) 1.59
Properties and ionic mechanisms of action potential adaptation, restitution, and accommodation in canine epicardium. Am J Physiol Heart Circ Physiol (2009) 1.56
Deranged sodium to sudden death. J Physiol (2015) 1.51
Transmural expression of transient outward potassium current subunits in normal and failing canine and human hearts. J Physiol (2004) 1.44
Remodeling in cells from different regions of the reentrant circuit during ventricular tachycardia. Circulation (2005) 1.42
Localization of the Kv1.5 K+ channel protein in explanted cardiac tissue. J Clin Invest (1995) 1.35
Role of activated CaMKII in abnormal calcium homeostasis and I(Na) remodeling after myocardial infarction: insights from mathematical modeling. J Mol Cell Cardiol (2008) 1.27
Dynamic remodeling of K+ and Ca2+ currents in cells that survived in the epicardial border zone of canine healed infarcted heart. Am J Physiol Heart Circ Physiol (2004) 1.22
Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density. Biophys J (2011) 1.22
Cyclosporine A attenuates mitochondrial permeability transition and improves mitochondrial respiratory function in cardiomyocytes isolated from dogs with heart failure. J Mol Cell Cardiol (2006) 1.21
EH domain proteins regulate cardiac membrane protein targeting. Circ Res (2010) 1.15
Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis. PLoS Comput Biol (2009) 1.14
Regulation of the ankyrin-B-based targeting pathway following myocardial infarction. Cardiovasc Res (2008) 1.12
Deleting the accessory subunit KChIP2 results in loss of I(to,f) and increased I(K,slow) that maintains normal action potential configuration. Heart Rhythm (2008) 1.04
Repolarization abnormalities and afterdepolarizations in a canine model of sudden cardiac death. Am J Physiol Regul Integr Comp Physiol (2008) 1.02
Ionic mechanisms of electrophysiological heterogeneity and conduction block in the infarct border zone. Am J Physiol Heart Circ Physiol (2010) 0.99
Diverse phenotypes of outward currents in cells that have survived in the 5-day-infarcted heart. Am J Physiol Heart Circ Physiol (2005) 0.97
Apamin-sensitive calcium-activated potassium currents in rabbit ventricles with chronic myocardial infarction. J Cardiovasc Electrophysiol (2013) 0.92
Extracellular space attenuates the effect of gap junctional remodeling on wave propagation: a computational study. Biophys J (2009) 0.92
Modulating L-type calcium current affects discontinuous cardiac action potential conduction. Biophys J (1996) 0.92
Targeting protein tyrosine phosphatase σ after myocardial infarction restores cardiac sympathetic innervation and prevents arrhythmias. Nat Commun (2015) 0.91
Inhibition of mitochondrial permeability transition pores by cyclosporine A improves cytochrome C oxidase function and increases rate of ATP synthesis in failing cardiomyocytes. Heart Fail Rev (2005) 0.91
Selective gamma-ketoaldehyde scavengers protect Nav1.5 from oxidant-induced inactivation. J Mol Cell Cardiol (2009) 0.87
The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarction. J Mol Cell Cardiol (2015) 0.87
Gene therapy for ventricular tachyarrhythmias. Gene Ther (2012) 0.82
Repolarization reserve evolves dynamically during the cardiac action potential: effects of transient outward currents on early afterdepolarizations. Circ Arrhythm Electrophysiol (2015) 0.79
Cardiac contractility modulation with the impulse dynamics signal: studies in dogs with chronic heart failure. Heart Fail Rev (2001) 0.79
Restoration of the transient outward potassium current by noradrenaline in chagasic canine epicardium. J Physiol (1997) 0.78
Ischemia-related subcellular redistribution of sodium channels enhances the proarrhythmic effect of class I antiarrhythmic drugs: a simulation study. PLoS One (2014) 0.77
Making better scar: Emerging approaches for modifying mechanical and electrical properties following infarction and ablation. Prog Biophys Mol Biol (2015) 0.77
Kvβ1.1 senses pyridine nucleotide changes in the mouse heart and modulates cardiac electrical activity. Am J Physiol Heart Circ Physiol (2016) 0.77
How does the shape of the cardiac action potential control calcium signaling and contraction in the heart? J Mol Cell Cardiol (2010) 0.76
Effect of skeletal muscle Na(+) channel delivered via a cell platform on cardiac conduction and arrhythmia induction. Circ Arrhythm Electrophysiol (2012) 0.76
Molecular Mechanisms of Sympathetic Remodeling and Arrhythmias. Circ Arrhythm Electrophysiol (2016) 0.76
Can heart function lost to disease be regenerated by therapeutic targeting of cardiac scar tissue? Semin Cell Dev Biol (2016) 0.75
Cardiac Sympathetic Nerve Sprouting and Susceptibility to Ventricular Arrhythmias after Myocardial Infarction. Cardiol Res Pract (2015) 0.75
Increased Short-Term Beat-To-Beat Variability of QT Interval in Patients with Acromegaly. PLoS One (2015) 0.75
Novel strategy to reduce sudden-death risk in the healing phase after myocardial infarction. Circulation (2008) 0.75
Impact of recent molecular studies on evaluation of ventricular arrhythmias. J Interv Card Electrophysiol (2000) 0.75
Pathophysiology and prevention of atrial fibrillation. Circulation (2001) 2.31
Delayed rectifier K currents have reduced amplitudes and altered kinetics in myocytes from infarcted canine ventricle. Cardiovasc Res (2000) 1.90
Alterations of Na+ currents in myocytes from epicardial border zone of the infarcted heart. A possible ionic mechanism for reduced excitability and postrepolarization refractoriness. Circ Res (1997) 1.65
Effects of mibefradil, a T-type calcium current antagonist, on electrophysiology of Purkinje fibers that survived in the infarcted canine heart. J Cardiovasc Electrophysiol (1999) 1.44
Electrical remodeling in ischemia and infarction. Cardiovasc Res (1999) 1.41
Age-related appearance of outward currents may contribute to developmental differences in ventricular repolarization. Circ Res (1992) 1.33
Multiple types of Ca2+ currents in single canine Purkinje cells. Circ Res (1989) 1.16
Electrical restitution process in dispersed canine cardiac Purkinje and ventricular cells. Am J Physiol (1987) 1.14
Different effects of intracellular Ca and protein kinase C on cardiac T and L Ca currents. Am J Physiol (1991) 1.12
Diminished Ca2+ and Ba2+ currents in myocytes surviving in the epicardial border zone of the 5-day infarcted canine heart. Circ Res (1995) 1.11
Ca2+ waves during triggered propagated contractions in intact trabeculae. Determinants of the velocity of propagation. Circ Res (1999) 1.07
Reduced inward rectifying and increased E-4031-sensitive K+ current density in arrhythmogenic subendocardial purkinje myocytes from the infarcted heart. J Cardiovasc Electrophysiol (1998) 1.00
Effects of left atrial enlargement on atrial transmembrane potentials and structure in dogs with mitral valve fibrosis. Am J Cardiol (1982) 0.99
Noradrenaline hyperpolarizes cells of the canine coronary sinus by increasing their permeability to potassium ions. J Physiol (1983) 0.98
Lidocaine action on Na+ currents in ventricular myocytes from the epicardial border zone of the infarcted heart. Circ Res (1998) 0.92
Calcium waves: physiological relevance in cardiac function. Circ Res (2000) 0.90
Ca2+ waves during triggered propagated contractions in intact trabeculae. Am J Physiol (1998) 0.90
Steady-state and nonsteady-state action potentials in fibrillating canine atrium: abnormal rate adaptation and its possible mechanisms. Cardiovasc Res (1999) 0.90
The basis for the membrane potential of quiescent cells of the canine coronary sinus. J Physiol (1983) 0.90
The effects on atrial electrophysiology and structure of surgically induced right atrial enlargement in dogs. Circ Res (1981) 0.90
Abnormalities in Ca(i)handling in myocytes that survive in the infarcted heart are not just due to alterations in repolarization. J Mol Cell Cardiol (2000) 0.89
Altered pharmacologic responsiveness of reduced L-type calcium currents in myocytes surviving in the infarcted heart. J Cardiovasc Electrophysiol (1996) 0.87
Characteristics of the beta-adrenergic receptor complex in the epicardial border zone of the 5-day infarcted canine heart. Circulation (1995) 0.86
Congestive heart failure after myocardial infarction in the rat: cardiac force and spontaneous sarcomere activity. Ann N Y Acad Sci (2004) 0.86
Ca(2+)-dependent outward currents in myocytes from epicardial border zone of 5-day infarcted canine heart. Am J Physiol (1997) 0.86
Circus movement in the canine atrium around the tricuspid ring during experimental atrial flutter and during reentry in vitro. Circulation (1987) 0.83
Effects of pharmacologic agents on induced atrial flutter in dogs with right atrial enlargement. J Cardiovasc Pharmacol (1986) 0.83
Mechanisms for atrial arrhythmias associated with cardiomyopathy: a study of feline hearts with primary myocardial disease. Circulation (1984) 0.82
Damage induced arrhythmias: mechanisms and implications. Can J Physiol Pharmacol (2001) 0.79
Effects of Bay Y5959 on Ca2+ currents and intracellular Ca2+ in cells that have survived in the epicardial border of the infarcted canine heart. J Cardiovasc Pharmacol (1999) 0.77
Three things you should know when considering the atria: location, location, location. Circ Res (2001) 0.75
Endothelin: AF-riend or AF-oe? J Cardiovasc Electrophysiol (2001) 0.75
Endocardial mapping of reentry around an anatomical barrier in the canine right atrium: observations during the action of the Class IC agent, flecainide. J Cardiovasc Electrophysiol (1993) 0.75