Interactions between cardiac cells enhance cardiomyocyte hypertrophy and increase fibroblast proliferation.

PubWeight™: 1.19‹?› | Rank: Top 10%

🔗 View Article (PMID 15389635)

Published in J Cell Physiol on March 01, 2005

Authors

Sandra Fredj1, Jocelyn Bescond, Claire Louault, Daniel Potreau

Author Affiliations

1: Laboratoire de Physiologie et Physiopathologie Cardiaques, UMR 6187 CNRS-Université de Poitiers, Poitiers, Cedex, France.

Articles citing this

Functional screening identifies miRNAs inducing cardiac regeneration. Nature (2012) 4.39

Cardiac fibroblast: the renaissance cell. Circ Res (2009) 4.01

The origin of fibroblasts and mechanism of cardiac fibrosis. J Cell Physiol (2010) 2.18

Cardiac fibroblast-derived microRNA passenger strand-enriched exosomes mediate cardiomyocyte hypertrophy. J Clin Invest (2014) 2.06

Dedifferentiation and proliferation of mammalian cardiomyocytes. PLoS One (2010) 1.89

Connective tissue growth factor promotes fibrosis downstream of TGFbeta and IL-6 in chronic cardiac allograft rejection. Am J Transplant (2009) 1.59

Cardiac Fibrosis: The Fibroblast Awakens. Circ Res (2016) 1.21

Klotho and phosphate are modulators of pathologic uremic cardiac remodeling. J Am Soc Nephrol (2014) 1.16

Critical role for IL-6 in hypertrophy and fibrosis in chronic cardiac allograft rejection. Am J Transplant (2009) 1.13

IL-6 loss causes ventricular dysfunction, fibrosis, reduced capillary density, and dramatically alters the cell populations of the developing and adult heart. Am J Physiol Heart Circ Physiol (2009) 1.11

Pathologic function and therapeutic potential of exosomes in cardiovascular disease. Biochim Biophys Acta (2014) 1.06

Immune cell and other noncardiomyocyte regulation of cardiac hypertrophy and remodeling. Circulation (2015) 0.96

TGF-beta, IL-6, IL-17 and CTGF direct multiple pathologies of chronic cardiac allograft rejection. Immunotherapy (2010) 0.92

Pathophysiological roles of FGF signaling in the heart. Front Physiol (2013) 0.87

Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends? J Cardiovasc Transl Res (2012) 0.86

Role of interleukin-6 in regulation of immune responses to remodeling after myocardial infarction. Heart Fail Rev (2015) 0.86

The Cardiomyocyte as a Source of Cytokines in Cardiac Injury. J Cell Sci Ther (2011) 0.84

More Than Tiny Sacks: Stem Cell Exosomes as Cell-Free Modality for Cardiac Repair. Circ Res (2016) 0.84

FGF21 and Cardiac Physiopathology. Front Endocrinol (Lausanne) (2015) 0.82

Exosomes: Fundamental Biology and Roles in Cardiovascular Physiology. Annu Rev Physiol (2015) 0.80

The origin and arrhythmogenic potential of fibroblasts in cardiac disease. J Cardiovasc Transl Res (2012) 0.80

Crosstalk between the heart and peripheral organs in heart failure. Exp Mol Med (2016) 0.77

A critical role of cardiac fibroblast-derived exosomes in activating renin angiotensin system in cardiomyocytes. J Mol Cell Cardiol (2015) 0.76

Small dedifferentiated cardiomyocytes bordering on microdomains of fibrosis: evidence for reverse remodeling with assisted recovery. J Cardiovasc Pharmacol (2014) 0.76

Thyroid Hormone Increases TGF-beta1 in Cardiomyocytes Cultures Independently of Angiotensin II Type 1 and Type 2 Receptors. Int J Endocrinol (2010) 0.75

Autonomous and Non-autonomous Defects Underlie Hypertrophic Cardiomyopathy in BRAF-Mutant hiPSC-Derived Cardiomyocytes. Stem Cell Reports (2016) 0.75

Myofibroblast secretome and its auto-/paracrine signaling. Expert Rev Cardiovasc Ther (2016) 0.75

Cardiac Dysfunction in Association with Increased Inflammatory Markers in Primary Aldosteronism. Endocrinol Metab (Seoul) (2016) 0.75

Exosomes as agents of change in the cardiovascular system. J Mol Cell Cardiol (2017) 0.75

Atrophied cardiomyocytes and their potential for rescue and recovery of ventricular function. Heart Fail Rev (2016) 0.75

Articles by these authors

Functional characterization of a Ca(2+)-activated non-selective cation channel in human atrial cardiomyocytes. J Physiol (2004) 1.22

Functional expression of the TRPM4 cationic current in ventricular cardiomyocytes from spontaneously hypertensive rats. Hypertension (2006) 1.10

Role of interleukin-6 in cardiomyocyte/cardiac fibroblast interactions during myocyte hypertrophy and fibroblast proliferation. J Cell Physiol (2005) 1.08

Secretion of IL-6, IL-11 and LIF by human cardiomyocytes in primary culture. Cytokine (2002) 0.96

Ion-channel mRNA-expression profiling: Insights into cardiac remodeling and arrhythmic substrates. J Mol Cell Cardiol (2009) 0.95

Human cardiomyocyte hypertrophy induced in vitro by gp130 stimulation. Cardiovasc Res (2003) 0.92

Molecular and functional characterization of a new potassium conductance in mouse ventricular fibroblasts. J Mol Cell Cardiol (2009) 0.90

Direct inhibition of the pacemaker (If) current in rabbit sinoatrial node cells by genistein. Br J Pharmacol (2006) 0.85

Implication of connexins 40 and 43 in functional coupling between mouse cardiac fibroblasts in primary culture. Biochim Biophys Acta (2008) 0.84

Rabbit, a relevant model for the study of cardiac beta 3-adrenoceptors. Exp Physiol (2009) 0.81

Molecular regulation and pharmacology of pacemaker channels. Curr Pharm Des (2007) 0.80

Calmodulin antagonist W7 directly inhibits f-type current in rabbit sino-atrial cells. Eur J Pharmacol (2005) 0.78

Brain natriuretic peptide secretion in adult rat heart muscle cells: the role of calcium channels. Arch Cardiovasc Dis (2008) 0.78

Depression of cardiac L-type calcium current by a scorpion venom fraction M1 following muscarinic receptors interaction involving adenylate cyclase pathway. Toxicon (2006) 0.77

Structure elucidation of a dihydropyranone from Tapinanthus dodoneifolius. J Nat Prod (2007) 0.75

Reexpression of the nifedipine-resistant calcium channel during dedifferentiation of adult rat ventricular cardiomyocytes. J Cardiovasc Electrophysiol (2002) 0.75

[Evidence for expression of a nifedipine resistant L-type calcium current in human atrial cardiomyocytes]. J Med Liban (2008) 0.75

Natural product hybrid and its superacid synthesized analogues: dodoneine and its derivatives show selective inhibition of carbonic anhydrase isoforms I, III, XIII and XIV. Bioorg Med Chem (2013) 0.75

A novel depolarizing activity of scorpion venom fraction M1 due to activation of skeletal muscle nicotinic receptors. Toxicon (2006) 0.75