|
1
|
Sudden death associated with short-QT syndrome linked to mutations in HERG.
|
Circulation
|
2003
|
3.68
|
|
2
|
Genetic and biophysical basis of sudden unexplained nocturnal death syndrome (SUNDS), a disease allelic to Brugada syndrome.
|
Hum Mol Genet
|
2002
|
2.16
|
|
3
|
Cryptic 5' splice site activation in SCN5A associated with Brugada syndrome.
|
J Mol Cell Cardiol
|
2004
|
1.81
|
|
4
|
Further insights into the effect of quinidine in short QT syndrome caused by a mutation in HERG.
|
J Cardiovasc Electrophysiol
|
2005
|
1.74
|
|
5
|
Phenotypic characterization of a large European family with Brugada syndrome displaying a sudden unexpected death syndrome mutation in SCN5A:.
|
J Cardiovasc Electrophysiol
|
2004
|
1.69
|
|
6
|
Genetic and biophysical basis for bupivacaine-induced ST segment elevation and VT/VF. Anesthesia unmasked Brugada syndrome.
|
Heart Rhythm
|
2006
|
1.56
|
|
7
|
About half of the late sodium current in cardiac myocytes from dog ventricle is due to non-cardiac-type Na(+) channels.
|
J Mol Cell Cardiol
|
2012
|
1.47
|
|
8
|
Molecular basis of species-specific expression of repolarizing K+ currents in the heart.
|
Am J Physiol Heart Circ Physiol
|
2003
|
1.27
|
|
9
|
Expression pattern of neuronal and skeletal muscle voltage-gated Na+ channels in the developing mouse heart.
|
J Physiol
|
2005
|
1.26
|
|
10
|
Short QT syndrome.
|
CMAJ
|
2005
|
1.17
|
|
11
|
Value of electrocardiographic parameters and ajmaline test in the diagnosis of Brugada syndrome caused by SCN5A mutations.
|
Circulation
|
2004
|
1.14
|
|
12
|
Compound heterozygous mutations P336L and I1660V in the human cardiac sodium channel associated with the Brugada syndrome.
|
Circulation
|
2006
|
1.08
|
|
13
|
Proven infection-related sepsis induces a differential stress response early after ICU admission.
|
Crit Care
|
2010
|
1.03
|
|
14
|
Lidocaine-induced Brugada syndrome phenotype linked to a novel double mutation in the cardiac sodium channel.
|
Circ Res
|
2008
|
1.00
|
|
15
|
Novel mutations in domain I of SCN5A cause Brugada syndrome.
|
Mol Genet Metab
|
2002
|
0.94
|
|
16
|
Identification of two domains involved in the assembly of transient receptor potential canonical channels.
|
J Biol Chem
|
2006
|
0.90
|
|
17
|
Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.
|
J Neuroinflammation
|
2014
|
0.88
|
|
18
|
Divergent action potential morphologies reveal nonequilibrium properties of human cardiac Na channels.
|
Cardiovasc Res
|
2004
|
0.87
|
|
19
|
Larger dispersion of INa in female dog ventricle as a mechanism for gender-specific incidence of cardiac arrhythmias.
|
Cardiovasc Res
|
2008
|
0.85
|
|
20
|
STIM1 participates in the contractile rhythmicity of HL-1 cells by moderating T-type Ca(2+) channel activity.
|
Biochim Biophys Acta
|
2013
|
0.83
|
|
21
|
C-Terminal modifications of apelin-13 significantly change ligand binding, receptor signaling, and hypotensive action.
|
J Med Chem
|
2015
|
0.79
|
|
22
|
Modulation of canine cardiac sodium current by Apelin.
|
J Mol Cell Cardiol
|
2009
|
0.79
|
|
23
|
ELABELA Improves Cardio-Renal Outcome in Fatal Experimental Septic Shock.
|
Crit Care Med
|
2017
|
0.75
|
|
24
|
Apelin Compared With Dobutamine Exerts Cardioprotection and Extends Survival in a Rat Model of Endotoxin-Induced Myocardial Dysfunction.
|
Crit Care Med
|
2017
|
0.75
|