|
1
|
Allelic variants in long-QT disease genes in patients with drug-associated torsades de pointes.
|
Circulation
|
2002
|
2.98
|
|
2
|
Mutation in glycerol-3-phosphate dehydrogenase 1 like gene (GPD1-L) decreases cardiac Na+ current and causes inherited arrhythmias.
|
Circulation
|
2007
|
2.50
|
|
3
|
Genetics of acquired long QT syndrome.
|
J Clin Invest
|
2005
|
1.96
|
|
4
|
Oxidative mediated lipid peroxidation recapitulates proarrhythmic effects on cardiac sodium channels.
|
Circ Res
|
2005
|
1.70
|
|
5
|
Clinical, genetic, and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block.
|
Circulation
|
2002
|
1.65
|
|
6
|
A sodium channel pore mutation causing Brugada syndrome.
|
Heart Rhythm
|
2006
|
1.64
|
|
7
|
Sex, age, and regional differences in L-type calcium current are important determinants of arrhythmia phenotype in rabbit hearts with drug-induced long QT type 2.
|
Circ Res
|
2008
|
1.30
|
|
8
|
Functional Interactions between Distinct Sodium Channel Cytoplasmic Domains through the Action of Calmodulin.
|
J Biol Chem
|
2009
|
1.22
|
|
9
|
New mechanism contributing to drug-induced arrhythmia: rescue of a misprocessed LQT3 mutant.
|
Circulation
|
2005
|
1.12
|
|
10
|
Quantitation of protein kinase A-mediated trafficking of cardiac sodium channels in living cells.
|
Cardiovasc Res
|
2006
|
1.07
|
|
11
|
Compound-specific Na+ channel pore conformational changes induced by local anaesthetics.
|
J Physiol
|
2005
|
0.87
|
|
12
|
HERG is protected from pharmacological block by alpha-1,2-glucosyltransferase function.
|
J Biol Chem
|
2006
|
0.85
|
|
13
|
Potential role of isoketals formed via the isoprostane pathway of lipid peroxidation in ischemic arrhythmias.
|
J Cardiovasc Pharmacol
|
2007
|
0.85
|