|
1
|
Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage.
|
Am J Physiol
|
1991
|
1.52
|
|
2
|
Polymicrobial sepsis but not low-dose endotoxin infusion causes decreased splenocyte IL-2/IFN-gamma release while increasing IL-4/IL-10 production.
|
J Surg Res
|
1994
|
1.36
|
|
3
|
Polymicrobial sepsis selectively activates peritoneal but not alveolar macrophages to release inflammatory mediators (interleukins-1 and -6 and tumor necrosis factor).
|
Circ Shock
|
1992
|
1.36
|
|
4
|
Hemorrhage induces an increase in serum TNF which is not associated with elevated levels of endotoxin.
|
Cytokine
|
1990
|
1.30
|
|
5
|
Hemorrhage induces enhanced Kupffer cell cytotoxicity while decreasing peritoneal or splenic macrophage capacity. Involvement of cell-associated tumor necrosis factor and reactive nitrogen.
|
J Immunol
|
1991
|
1.11
|
|
6
|
Differential effects of hemorrhage on Kupffer cells: decreased antigen presentation despite increased inflammatory cytokine (IL-1, IL-6 and TNF) release.
|
Cytokine
|
1992
|
1.07
|
|
7
|
Role of interleukin 6 and transforming growth factor-beta in the induction of depressed splenocyte responses following sepsis.
|
Arch Surg
|
1993
|
0.95
|
|
8
|
Does endotoxin tolerance prevent the release of inflammatory monokines (interleukin 1, interleukin 6, or tumor necrosis factor) during sepsis?
|
Arch Surg
|
1992
|
0.87
|
|
9
|
Sepsis induces an early increased spontaneous release of hepatocellular stimulatory factor (interleukin-6) by Kupffer cells in both endotoxin tolerant and intolerant mice.
|
J Surg Res
|
1992
|
0.80
|
|
10
|
Insights into the mechanism by which interferon-gamma improves macrophage function following hemorrhage and resuscitation.
|
J Surg Res
|
1993
|
0.75
|