Published in Circ Shock on April 01, 1992
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Mechanism of immunosuppression in males following trauma-hemorrhage. Critical role of testosterone. Arch Surg (1996) 1.17
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17 beta-Estradiol normalizes immune responses in ovariectomized females after trauma-hemorrhage. Am J Physiol Cell Physiol (2001) 1.16
The induction of accelerated thymic programmed cell death during polymicrobial sepsis: control by corticosteroids but not tumor necrosis factor. Shock (1995) 1.14
The aromatase inhibitor, 4-hydroxyandrostenedione, restores immune responses following trauma-hemorrhage in males and decreases mortality from subsequent sepsis. Shock (2000) 1.12
IL-10 mediation of activation-induced TH1 cell apoptosis and lymphoid dysfunction in polymicrobial sepsis. Cytokine (2001) 1.11
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Up-regulation of a novel potent vasodilatory peptide adrenomedullin during polymicrobial sepsis. Shock (1998) 1.06
Testosterone receptor blockade after trauma-hemorrhage improves cardiac and hepatic functions in males. Am J Physiol (1997) 1.05
Testosterone: the crucial hormone responsible for depressing myocardial function in males after trauma-hemorrhage. Ann Surg (1998) 1.05
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Male sex steroids are responsible for depressing macrophage immune function after trauma-hemorrhage. Am J Physiol (1997) 1.05
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Flutamide: a novel agent for restoring the depressed cell-mediated immunity following soft-tissue trauma and hemorrhagic shock. Shock (1997) 1.04
Insight into the mechanism by which estradiol improves organ functions after trauma-hemorrhage. Surgery (2000) 1.03
Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation. Am J Physiol (1993) 1.03
Insights into the role of gammadelta T lymphocytes in the immunopathogenic response to thermal injury. J Leukoc Biol (2000) 1.02
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Inhibition of Fas signaling prevents hepatic injury and improves organ blood flow during sepsis. Surgery (2001) 1.02
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Sustained elevation in circulating catecholamine levels during polymicrobial sepsis. Shock (1995) 1.00
Role of Kupffer cells in interleukin-6 release following trauma-hemorrhage and resuscitation. Shock (1994) 0.99
Does endotoxin play a major role in inducing the depression of macrophage function during polymicrobial sepsis? Arch Surg (1995) 0.99
Differential alterations in systemic and regional oxygen delivery and consumption during the early and late stages of sepsis. J Trauma (1999) 0.99
Evidence for the uptake of ATP by rat soleus muscle in vitro. Biochim Biophys Acta (1970) 0.99
Chloroquine attenuates hemorrhagic shock-induced immunosuppression and decreases susceptibility to sepsis. Arch Surg (1992) 0.99
Does Fas ligand or endotoxin contribute to thymic apoptosis during polymicrobial sepsis? Shock (1999) 0.97
Do female sex steroids adversely or beneficially affect the depressed immune responses in males after trauma-hemorrhage? Arch Surg (2000) 0.97
Evolution of an immune suppressive macrophage phenotype as a product of P38 MAPK activation in polymicrobial sepsis. Shock (2001) 0.97