PubRank

Jialing Lin

Author PubWeight™ 23.31‹?›

Top papers

Rank Title Journal Year PubWeight™‹?›
1 Embedded together: the life and death consequences of interaction of the Bcl-2 family with membranes. Apoptosis 2007 2.82
2 Bcl-2 changes conformation to inhibit Bax oligomerization. EMBO J 2006 1.97
3 Signal recognition particle binds to ribosome-bound signal sequences with fluorescence-detected subnanomolar affinity that does not diminish as the nascent chain lengthens. J Biol Chem 2003 1.71
4 A short Nur77-derived peptide converts Bcl-2 from a protector to a killer. Cancer Cell 2008 1.66
5 Auto-activation of the apoptosis protein Bax increases mitochondrial membrane permeability and is inhibited by Bcl-2. J Biol Chem 2006 1.65
6 Cotranslational protein integration into the ER membrane is mediated by the binding of nascent chains to translocon proteins. Mol Cell 2003 1.44
7 Bax forms an oligomer via separate, yet interdependent, surfaces. J Biol Chem 2010 1.27
8 Bcl-2 homodimerization involves two distinct binding surfaces, a topographic arrangement that provides an effective mechanism for Bcl-2 to capture activated Bax. J Biol Chem 2004 1.20
9 tBid elicits a conformational alteration in membrane-bound Bcl-2 such that it inhibits Bax pore formation. J Biol Chem 2006 1.15
10 Bcl-2 and Bax interact via the BH1-3 groove-BH3 motif interface and a novel interface involving the BH4 motif. J Biol Chem 2010 1.10
11 After embedding in membranes antiapoptotic Bcl-XL protein binds both Bcl-2 homology region 3 and helix 1 of proapoptotic Bax protein to inhibit apoptotic mitochondrial permeabilization. J Biol Chem 2014 0.97
12 sHA 14-1, a stable and ROS-free antagonist against anti-apoptotic Bcl-2 proteins, bypasses drug resistances and synergizes cancer therapies in human leukemia cell. Cancer Lett 2007 0.91
13 Natural diterpenoid compound elevates expression of Bim protein, which interacts with antiapoptotic protein Bcl-2, converting it to proapoptotic Bax-like molecule. J Biol Chem 2011 0.90
14 Oligomerization of membrane-bound Bcl-2 is involved in its pore formation induced by tBid. Apoptosis 2009 0.89
15 Amphipathic tail-anchoring peptide and Bcl-2 homology domain-3 (BH3) peptides from Bcl-2 family proteins induce apoptosis through different mechanisms. J Biol Chem 2010 0.86
16 Hyaluronan synthase mediates dye translocation across liposomal membranes. BMC Biochem 2012 0.84
17 The cytosolic domain of Bcl-2 forms small pores in model mitochondrial outer membrane after acidic pH-induced membrane association. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi 2009 0.80
18 The cytosolic domain of Bcl-2 oligomerizes to form pores in model mitochondrial outer membrane at acidic pH. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi 2009 0.76
19 The Bax BH3 peptide H2-H3 promotes apoptosis by inhibiting Bcl-2's pore-forming and anti-Bax activities in the membrane. Sheng Wu Yi Xue Gong Cheng Xue Za Zhi 2009 0.75