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Randall C Johnson
Author PubWeight™ 33.47
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Top papers
Rank
Title
Journal
Year
PubWeight™
‹?›
1
MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis.
Nat Genet
2008
9.00
2
APOL1 genetic variants in focal segmental glomerulosclerosis and HIV-associated nephropathy.
J Am Soc Nephrol
2011
4.80
3
The high-frequency major histocompatibility complex class I allele Mamu-B*17 is associated with control of simian immunodeficiency virus SIVmac239 replication.
J Virol
2006
4.26
4
Human immunodeficiency virus type 1-specific CD8+ T-cell responses during primary infection are major determinants of the viral set point and loss of CD4+ T cells.
J Virol
2009
2.51
5
Dense mapping of MYH9 localizes the strongest kidney disease associations to the region of introns 13 to 15.
Hum Mol Genet
2010
2.51
6
Differential effects of MYH9 and APOL1 risk variants on FRMD3 Association with Diabetic ESRD in African Americans.
PLoS Genet
2011
1.57
7
MHC heterozygote advantage in simian immunodeficiency virus-infected Mauritian cynomolgus macaques.
Sci Transl Med
2010
1.38
8
Evaluation of nonviral risk factors for nasopharyngeal carcinoma in a high-risk population of Southern China.
Int J Cancer
2009
1.32
9
Genome-wide association study implicates PARD3B-based AIDS restriction.
J Infect Dis
2011
1.15
10
Host genetic influences on highly active antiretroviral therapy efficacy and AIDS-free survival.
J Acquir Immune Defic Syndr
2008
1.09
11
Genetic factors leading to chronic Epstein-Barr virus infection and nasopharyngeal carcinoma in South East China: study design, methods and feasibility.
Hum Genomics
2006
1.01
12
Pathological manifestations of feline immunodeficiency virus (FIV) infection in wild African lions.
Virology
2009
0.95
13
Consistent effects of TSG101 genetic variability on multiple outcomes of exposure to human immunodeficiency virus type 1.
J Virol
2006
0.92
14
Acute-phase CD8 T cell responses that select for escape variants are needed to control live attenuated simian immunodeficiency virus.
J Virol
2013
0.87
15
Activating killer-cell immunoglobulin-like receptors (KIR) and their cognate HLA ligands are significantly increased in autism.
Brain Behav Immun
2012
0.82