1
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Changing patterns of Plasmodium blood-stage infections in the Wosera region of Papua New Guinea monitored by light microscopy and high throughput PCR diagnosis.
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Am J Trop Med Hyg
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2006
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1.93
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2
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Differential patterns of infection and disease with P. falciparum and P. vivax in young Papua New Guinean children.
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PLoS One
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2010
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1.87
|
3
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Comparison of Plasmodium falciparum allelic frequency distribution in different endemic settings by high-resolution genotyping.
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Malar J
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2009
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1.67
|
4
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High sensitivity detection of Plasmodium species reveals positive correlations between infections of different species, shifts in age distribution and reduced local variation in Papua New Guinea.
|
Malar J
|
2009
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1.62
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5
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Features and prognosis of severe malaria caused by Plasmodium falciparum, Plasmodium vivax and mixed Plasmodium species in Papua New Guinean children.
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PLoS One
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2011
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1.55
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6
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Multiplicity and diversity of Plasmodium vivax infections in a highly endemic region in Papua New Guinea.
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PLoS Negl Trop Dis
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2011
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1.47
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7
|
Multilocus haplotypes reveal variable levels of diversity and population structure of Plasmodium falciparum in Papua New Guinea, a region of intense perennial transmission.
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Malar J
|
2010
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1.47
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8
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Reduced Plasmodium vivax erythrocyte infection in PNG Duffy-negative heterozygotes.
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PLoS One
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2007
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1.45
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9
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Force of infection is key to understanding the epidemiology of Plasmodium falciparum malaria in Papua New Guinean children.
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Proc Natl Acad Sci U S A
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2012
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1.33
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10
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Mass drug administration trial to eliminate lymphatic filariasis in Papua New Guinea: changes in microfilaremia, filarial antigen, and Bm14 antibody after cessation.
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Am J Trop Med Hyg
|
2008
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1.25
|
11
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How much remains undetected? Probability of molecular detection of human Plasmodia in the field.
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PLoS One
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2011
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1.23
|
12
|
Relapses contribute significantly to the risk of Plasmodium vivax infection and disease in Papua New Guinean children 1-5 years of age.
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J Infect Dis
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2012
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1.23
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13
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Three different Plasmodium species show similar patterns of clinical tolerance of malaria infection.
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Malar J
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2009
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1.09
|
14
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A high force of plasmodium vivax blood-stage infection drives the rapid acquisition of immunity in papua new guinean children.
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PLoS Negl Trop Dis
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2013
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1.04
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15
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Treatment with coartem (artemether-lumefantrine) in Papua New Guinea.
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Am J Trop Med Hyg
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2010
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0.98
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16
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High levels of genetic diversity of Plasmodium falciparum populations in Papua New Guinea despite variable infection prevalence.
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Am J Trop Med Hyg
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2013
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0.96
|
17
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Naturally acquired immune responses to P. vivax merozoite surface protein 3α and merozoite surface protein 9 are associated with reduced risk of P. vivax malaria in young Papua New Guinean children.
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PLoS Negl Trop Dis
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2013
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0.95
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18
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Reduction in acute filariasis morbidity during a mass drug administration trial to eliminate lymphatic filariasis in Papua New Guinea.
|
PLoS Negl Trop Dis
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2011
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0.86
|
19
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Tolerability and safety of primaquine in Papua New Guinean children 1 to 10 years of age.
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Antimicrob Agents Chemother
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2012
|
0.83
|
20
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Seroprevalence of antibodies to parvovirus B19 among children in Papua New Guinea.
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Am J Trop Med Hyg
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2007
|
0.79
|