1
|
Mice lacking ZFHX1B, the gene that codes for Smad-interacting protein-1, reveal a role for multiple neural crest cell defects in the etiology of Hirschsprung disease-mental retardation syndrome.
|
Am J Hum Genet
|
2003
|
1.88
|
2
|
Generation of the floxed allele of the SIP1 (Smad-interacting protein 1) gene for Cre-mediated conditional knockout in the mouse.
|
Genesis
|
2002
|
1.34
|
3
|
Ttrap is an essential modulator of Smad3-dependent Nodal signaling during zebrafish gastrulation and left-right axis determination.
|
Development
|
2007
|
1.29
|
4
|
Interaction between Smad-interacting protein-1 and the corepressor C-terminal binding protein is dispensable for transcriptional repression of E-cadherin.
|
J Biol Chem
|
2003
|
1.22
|
5
|
Preclinical characterization of GLPG0634, a selective inhibitor of JAK1, for the treatment of inflammatory diseases.
|
J Immunol
|
2013
|
1.07
|
6
|
Neural crest-specific removal of Zfhx1b in mouse leads to a wide range of neurocristopathies reminiscent of Mowat-Wilson syndrome.
|
Hum Mol Genet
|
2007
|
0.93
|
7
|
Discovery and optimization of an azetidine chemical series as a free fatty acid receptor 2 (FFA2) antagonist: from hit to clinic.
|
J Med Chem
|
2014
|
0.85
|
8
|
Cloning and expression of the cyclophilin Bet v 7, and analysis of immunological cross-reactivity among the cyclophilin A family.
|
Mol Immunol
|
2005
|
0.81
|
9
|
Essential validation of gene trap mouse ES cell lines: a test case with the gene Ttrap.
|
Int J Dev Biol
|
2009
|
0.80
|
10
|
Smicl is a novel Smad interacting protein and cleavage and polyadenylation specificity factor associated protein.
|
Genes Cells
|
2005
|
0.80
|
11
|
Triazolopyridines as selective JAK1 inhibitors: from hit identification to GLPG0634.
|
J Med Chem
|
2014
|
0.76
|