Moesin helps to restrain synaptic growth at the Drosophila neuromuscular junction.

PubWeight™: 0.85‹?›

🔗 View Article (PMID 18161855)

Published in Dev Neurobiol on February 15, 2008

Authors

Sara Seabrooke1, Bryan A Stewart

Author Affiliations

1: Department of Biology, University of Toronto, Mississauga, Ontario, Canada L5L 1C6.

Articles by these authors

Members of the synaptobrevin/vesicle-associated membrane protein (VAMP) family in Drosophila are functionally interchangeable in vivo for neurotransmitter release and cell viability. Proc Natl Acad Sci U S A (2002) 1.06

A genetic screen for suppressors of Drosophila NSF2 neuromuscular junction overgrowth. Genetics (2005) 1.05

Dystroglycan and protein O-mannosyltransferases 1 and 2 are required to maintain integrity of Drosophila larval muscles. Mol Biol Cell (2007) 1.03

A Drosophila SNAP-25 null mutant reveals context-dependent redundancy with SNAP-24 in neurotransmission. Genetics (2002) 0.97

Synaptic vesicle mobility and presynaptic F-actin are disrupted in a N-ethylmaleimide-sensitive factor allele of Drosophila. Mol Biol Cell (2006) 0.94

Nonmuscle Myosin II helps regulate synaptic vesicle mobility at the Drosophila neuromuscular junction. BMC Neurosci (2010) 0.90

Functional roles for beta1,4-N-acetlygalactosaminyltransferase-A in Drosophila larval neurons and muscles. Genetics (2006) 0.89

Synaptic transmission and plasticity are modulated by nonmuscle myosin II at the neuromuscular junction of Drosophila. J Neurophysiol (2011) 0.88

Myosin VI contributes to synaptic transmission and development at the Drosophila neuromuscular junction. BMC Neurosci (2011) 0.85

Interaction of cytoskeleton genes with NSF2-induced neuromuscular junction overgrowth. Genesis (2006) 0.82

Effect of juxtamembrane tryptophans on the immersion depth of Synaptobrevin, an integral vesicle membrane protein. Biochim Biophys Acta (2012) 0.78