Published in Shock on December 01, 2008
Activation of toll-like receptors and inflammasome complexes in the diabetic cardiomyopathy-associated inflammation. Int J Endocrinol (2014) 0.85
Microglial CD14 activated by iNOS contributes to neuroinflammation in cerebral ischemia. Brain Res (2013) 0.84
Cardiovascular Dysfunction Following Burn Injury: What We Have Learned from Rat and Mouse Models. Int J Mol Sci (2016) 0.82
Commensal microflora induce host defense and decrease bacterial translocation in burn mice through toll-like receptor 4. J Biomed Sci (2010) 0.80
Burn serum causes a CD14-dependent mitochondrial damage in primary cardiomyocytes. Am J Physiol Heart Circ Physiol (2010) 0.79
Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium. Purinergic Signal (2016) 0.75
American Burn Association consensus conference to define sepsis and infection in burns. J Burn Care Res (2007) 2.71
Tumor necrosis factor-alpha-induced caspase activation mediates endotoxin-related cardiac dysfunction. Crit Care Med (2005) 1.73
Sodium/hydrogen exchange activity in sepsis and in sepsis complicated by previous injury: 31P and 23Na NMR study. Crit Care Med (2005) 1.46
Protective role of heat stress in burn trauma. Crit Care Med (2004) 1.41
17β-Estradiol reappropriates mass lost to the hypermetabolic state in thermally injured rats. J Surg Res (2012) 1.41
Nitric oxide donors alter cardiomyocyte cytokine secretion and cardiac function. Crit Care Med (2005) 1.41
Recombinant plasma gelsolin infusion attenuates burn-induced pulmonary microvascular dysfunction. J Appl Physiol (1985) (2003) 1.28
IL-1beta and IL-6 act synergistically with TNF-alpha to alter cardiac contractile function after burn trauma. Shock (2002) 1.22
Macrophage migration inhibitory factor is a cardiac-derived myocardial depressant factor. Am J Physiol Heart Circ Physiol (2003) 1.19
Endotoxin-induced cardiomyopathy and systemic inflammation in mice is prevented by aldose reductase inhibition. Circulation (2006) 1.17
Macrophage migration inhibitory factor mediates late cardiac dysfunction after burn injury. Am J Physiol Heart Circ Physiol (2004) 1.09
Innate immunity SNPs are associated with risk for severe sepsis after burn injury. Clin Med Res (2006) 1.06
Role of interleukin-6 in cardiac inflammation and dysfunction after burn complicated by sepsis. Am J Physiol Heart Circ Physiol (2007) 1.05
IRAK1 deletion disrupts cardiac Toll/IL-1 signaling and protects against contractile dysfunction. Am J Physiol Heart Circ Physiol (2003) 0.98
Cardiac mitochondrial damage and loss of ROS defense after burn injury: the beneficial effects of antioxidant therapy. J Appl Physiol (1985) (2006) 0.97
Burn-induced impairment of cardiac contractile function is due to gut-derived factors transported in mesenteric lymph. Shock (2002) 0.97
Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves cardiac function in a rat pneumonia-related sepsis model. Am J Physiol Heart Circ Physiol (2012) 0.96
Bone marrow-derived cells contribute to contractile dysfunction in endotoxic shock. Am J Physiol Heart Circ Physiol (2004) 0.96
Activation of stress-responsive pathways by the sympathetic nervous system in burn trauma. Shock (2002) 0.93
Cone visual pigments of the Australian marsupials, the stripe-faced and fat-tailed dunnarts: sequence and inferred spectral properties. Vis Neurosci (2004) 0.93
Cardiac molecular signaling after burn trauma. J Burn Care Res (2006) 0.92
Gender-related differences in myocardial inflammatory and contractile responses to major burn trauma. Am J Physiol Heart Circ Physiol (2003) 0.90
Increasing percent burn is correlated with increasing inflammation in an adult rodent model. Shock (2008) 0.89
Cardiac mitochondrial damage and inflammation responses in sepsis. Surg Infect (Larchmt) (2007) 0.89
Estrogen treatment following severe burn injury reduces brain inflammation and apoptotic signaling. J Neuroinflammation (2009) 0.87
I kappa B overexpression in cardiomyocytes prevents NF-kappa B translocation and provides cardioprotection in trauma. Am J Physiol Heart Circ Physiol (2003) 0.87
Cardiac effects of burn injury complicated by aspiration pneumonia-induced sepsis. Am J Physiol Heart Circ Physiol (2003) 0.87
CD14-159 C allele is associated with increased risk of mortality after burn injury. Shock (2007) 0.86
Burn trauma alters calcium transporter protein expression in the heart. J Appl Physiol (1985) (2004) 0.86
Cardiomyocyte intracellular calcium and cardiac dysfunction after burn trauma. Crit Care Med (2002) 0.86
Estrogen-provided cardiac protection following burn trauma is mediated through a reduction in mitochondria-derived DAMPs. Am J Physiol Heart Circ Physiol (2014) 0.85
Hypertonic saline dextran after burn injury decreases inflammatory cytokine responses to subsequent pneumonia-related sepsis. Am J Physiol Heart Circ Physiol (2005) 0.84
Murine in vivo myocardial contractile dysfunction after burn injury is exacerbated by pneumonia sepsis. Shock (2005) 0.84
Association of mitochondrial allele 4216C with increased risk for sepsis-related organ dysfunction and shock after burn injury. Shock (2010) 0.83
TLR4 inactivation and rBPI(21) block burn-induced myocardial contractile dysfunction. Am J Physiol Heart Circ Physiol (2002) 0.83
Cardiac myocyte accumulation of calcium in burn injury: cause or effect of myocardial contractile dysfunction. J Burn Care Rehabil (2005) 0.82
Age-dependent differences of interleukin-6 activity in cardiac function after burn complicated by sepsis. Burns (2009) 0.82
Rho-associated kinase modulates myocardial inflammatory cytokine responses. Shock (2005) 0.82
Effects of burn injury on myocardial signaling and cytokine secretion: Possible role of PKC. Am J Physiol Regul Integr Comp Physiol (2006) 0.81
Caspase inhibition reduces cardiac myocyte dyshomeostasis and improves cardiac contractile function after major burn injury. J Appl Physiol (1985) (2007) 0.81
Molecular and pharmacological approaches to inhibiting nitric oxide after burn trauma. Am J Physiol Heart Circ Physiol (2003) 0.80
Echocardiography assessment of myocardial function after burn injury. Shock (2006) 0.80
Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses. Am J Physiol Heart Circ Physiol (2006) 0.80
Myocardial inflammatory responses to sepsis complicated by previous burn injury. Surg Infect (Larchmt) (2003) 0.80
Effect of thermal injury on relative anaplerosis and gluconeogenesis in the rat during infusion of [U-13C] propionate. Burns (2002) 0.80
IRAK contributes to burn-triggered myocardial contractile dysfunction. Am J Physiol Heart Circ Physiol (2002) 0.80
Epistatic interactions are critical to gene-association studies: PAI-1 and risk for mortality after burn injury. J Burn Care Res (2008) 0.79
Burn serum causes a CD14-dependent mitochondrial damage in primary cardiomyocytes. Am J Physiol Heart Circ Physiol (2010) 0.79
Effects of burn serum on myocardial inflammation and function. Shock (2004) 0.79
Sepsis-induced cardiac mitochondrial dysfunction involves altered mitochondrial-localization of tyrosine kinase Src and tyrosine phosphatase SHP2. PLoS One (2012) 0.79
Selective decontamination of the digestive tract attenuated the myocardial inflammation and dysfunction that occur with burn injury. Am J Physiol Heart Circ Physiol (2004) 0.79
Bactericidal/permeability increasing protein attenuates the myocardial inflammation/dysfunction that occurs with burn complicated by subsequent infection. J Appl Physiol (1985) (2007) 0.78
Sepsis and burn complicated by sepsis alter cardiac transporter expression. Burns (2006) 0.78
Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function. Am J Physiol Heart Circ Physiol (2004) 0.77
Reducing susceptibility to bacteremia after experimental burn injury: a role for selective decontamination of the digestive tract. J Appl Physiol (1985) (2007) 0.77
The effects of early excision and grafting on myocardial inflammation and function after burn injury. J Trauma (2006) 0.77
Sympathoadrenal modulation of stress-activated signaling in burn trauma. J Burn Care Rehabil (2002) 0.76
Heart Rate Variability as a Predictor of Death in Burn Patients. J Burn Care Res (2016) 0.75
Burn injury decreases myocardial Na-K-ATPase activity: role of PKC inhibition. Am J Physiol Regul Integr Comp Physiol (2007) 0.75