Contractile dysfunctions in ATP-dependent K+ channel-deficient mouse muscle during fatigue involve excessive depolarization and Ca2+ influx through L-type Ca2+ channels.

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Published in Exp Physiol on June 27, 2008

Authors

Carlo Cifelli1, Louise Boudreault, Bing Gong, Jean-Philippe Bercier, Jean-Marc Renaud

Author Affiliations

1: University of Ottawa, Department of Cellular and Molecular Medicine, 451 Smyth Road, Ottawa, Ontario, Canada K1H 8M5.

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