Published in Hypertension on July 07, 2008
Matrix metalloproteinases cleave the beta2-adrenergic receptor in spontaneously hypertensive rats. Am J Physiol Heart Circ Physiol (2010) 1.22
Matrix metalloproteinases: discrete elevations in essential hypertension and hypertensive end-stage renal disease. Clin Exp Hypertens (2009) 1.20
Enhanced matrix metalloproteinase activity in the spontaneously hypertensive rat: VEGFR-2 cleavage, endothelial apoptosis, and capillary rarefaction. J Vasc Res (2010) 1.14
The endothelial glycocalyx as a barrier to leukocyte adhesion and its mediation by extracellular proteases. Ann Biomed Eng (2011) 1.06
Receptor cleavage reduces the fluid shear response in neutrophils of the spontaneously hypertensive rat. Am J Physiol Cell Physiol (2010) 0.97
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Protease activity increases in plasma, peritoneal fluid, and vital organs after hemorrhagic shock in rats. PLoS One (2012) 0.92
2008 Landis Award lecture. Inflammation and the autodigestion hypothesis. Microcirculation (2009) 0.91
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Relative roles of doxycycline and cation chelation in endothelial glycan shedding and adhesion of leukocytes. Am J Physiol Heart Circ Physiol (2010) 0.88
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Pancreatic digestive enzyme blockade in the small intestine prevents insulin resistance in hemorrhagic shock. Shock (2014) 0.87
Matrix metalloproteinase activity causes VEGFR-2 cleavage and microvascular rarefaction in rat mesentery. Microcirculation (2011) 0.85
Constitutive expression and enzymatic cleavage of ICAM-1 in the spontaneously hypertensive rat. J Vasc Res (2011) 0.84
Acute venous occlusion enhances matrix metalloprotease activity: Implications on endothelial dysfunction. Microvasc Res (2010) 0.83
Nuclear factor kappa B and matrix metalloproteinase induced receptor cleavage in the spontaneously hypertensive rat. Hypertension (2011) 0.82
An emerging role of degrading proteinases in hypertension and the metabolic syndrome: autodigestion and receptor cleavage. Curr Hypertens Rep (2012) 0.81
Proteolytic Cleavage of the Red Blood Cell Glycocalyx in a Genetic Form of Hypertension. Cell Mol Bioeng (2011) 0.81
Receptor cleavage and P-selectin-dependent reduction of leukocyte adhesion in the spontaneously hypertensive rat. J Leukoc Biol (2012) 0.81
Detection of protease activities by flash chronopotentiometry using a reversible polycation-sensitive polymeric membrane electrode. Anal Biochem (2011) 0.80
Proof of Concept: Matrix metalloproteinase inhibitor decreases inflammation and improves muscle insulin sensitivity in people with type 2 diabetes. J Inflamm (Lond) (2012) 0.79
The autodigestion hypothesis for shock and multi-organ failure. Ann Biomed Eng (2013) 0.78
Proteolytic Activity Attenuates the Response of Endothelial Cells to Fluid Shear Stress. Cell Mol Bioeng (2012) 0.78
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The oxygen free radicals control MMP-9 and transcription factors expression in the spontaneously hypertensive rat. Microvasc Res (2013) 0.76
Cleavage and reduced CD36 ectodomain density on heart and spleen macrophages in the spontaneously hypertensive rat. Microvasc Res (2014) 0.76
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Metalloproteinases damage the insulin receptor to cause insulin resistance in spontaneously hypertensive rats. Hypertension (2008) 0.75
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Enteral tranexamic acid attenuates vasopressor resistance and changes in α1-adrenergic receptor expression in hemorrhagic shock. J Trauma Acute Care Surg (2017) 0.75
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Matrix metalloproteinases cleave the beta2-adrenergic receptor in spontaneously hypertensive rats. Am J Physiol Heart Circ Physiol (2010) 1.22
Matrix metalloproteinases: discrete elevations in essential hypertension and hypertensive end-stage renal disease. Clin Exp Hypertens (2009) 1.20
The inflammatory aspect of the microcirculation in hypertension: oxidative stress, leukocytes/endothelial interaction, apoptosis. Microcirculation (2002) 1.19
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Enhanced matrix metalloproteinase activity in the spontaneously hypertensive rat: VEGFR-2 cleavage, endothelial apoptosis, and capillary rarefaction. J Vasc Res (2010) 1.14
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Pancreatic digestive enzyme blockade in the intestine increases survival after experimental shock. Sci Transl Med (2013) 1.12
Oxidative stress promotes endothelial cell apoptosis and loss of microvessels in the spontaneously hypertensive rats. Arterioscler Thromb Vasc Biol (2005) 1.11
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Receptor cleavage reduces the fluid shear response in neutrophils of the spontaneously hypertensive rat. Am J Physiol Cell Physiol (2010) 0.97
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Microvascular display of xanthine oxidase and NADPH oxidase in the spontaneously hypertensive rat. Microcirculation (2006) 0.93
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An in-vivo analysis of capillary stasis and endothelial apoptosis in a model of hypertension. Microcirculation (2008) 0.92
An animal model of venous hypertension: the role of inflammation in venous valve failure. J Vasc Surg (2005) 0.92
Protease activity increases in plasma, peritoneal fluid, and vital organs after hemorrhagic shock in rats. PLoS One (2012) 0.92
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Removal of luminal content protects the small intestine during hemorrhagic shock but is not sufficient to prevent lung injury. Physiol Rep (2013) 0.91
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Xanthine oxidase activity in the dexamethasone-induced hypertensive rat. Microvasc Res (2003) 0.89
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage. Expert Rev Endocrinol Metab (2010) 0.89
Intraluminal pancreatic serine protease activity, mucosal permeability, and shock: a review. Shock (2006) 0.89
Hypertension-induced venous valve remodeling. J Vasc Surg (2004) 0.89
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Pancreatic digestive enzyme blockade in the small intestine prevents insulin resistance in hemorrhagic shock. Shock (2014) 0.87
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An electrophoretic method for the detection of chymotrypsin and trypsin activity directly in whole blood. Electrophoresis (2010) 0.86
Autodigestion: Proteolytic Degradation and Multiple Organ Failure in Shock. Shock (2016) 0.85
Matrix metalloproteinase activity causes VEGFR-2 cleavage and microvascular rarefaction in rat mesentery. Microcirculation (2011) 0.85
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Constitutive expression and enzymatic cleavage of ICAM-1 in the spontaneously hypertensive rat. J Vasc Res (2011) 0.84
Acute venous occlusion enhances matrix metalloprotease activity: Implications on endothelial dysfunction. Microvasc Res (2010) 0.83
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Nuclear factor kappa B and matrix metalloproteinase induced receptor cleavage in the spontaneously hypertensive rat. Hypertension (2011) 0.82
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Receptor cleavage and P-selectin-dependent reduction of leukocyte adhesion in the spontaneously hypertensive rat. J Leukoc Biol (2012) 0.81
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Proteolytic Cleavage of the Red Blood Cell Glycocalyx in a Genetic Form of Hypertension. Cell Mol Bioeng (2011) 0.81
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Proteolytic Activity Attenuates the Response of Endothelial Cells to Fluid Shear Stress. Cell Mol Bioeng (2012) 0.78
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Intrinsic disturbance of cellular redox balance enhances blood lymphocyte apoptosis in the spontaneously hypertensive rat. Free Radic Biol Med (2006) 0.77
Enhancement of glucocorticoid and mineralocorticoid receptor density in the microcirculation of the spontaneously hypertensive rat. Microcirculation (2004) 0.77
Whole blood assay for trypsin activity using polyanionic focusing gel electrophoresis. Electrophoresis (2010) 0.76
The oxygen free radicals control MMP-9 and transcription factors expression in the spontaneously hypertensive rat. Microvasc Res (2013) 0.76
Matrix metalloproteinases activities in hypertension: emerging opportunities. Hypertension (2010) 0.76
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Enteral tranexamic acid attenuates vasopressor resistance and changes in α1-adrenergic receptor expression in hemorrhagic shock. J Trauma Acute Care Surg (2017) 0.75