1
|
Selection of genetic markers for association analyses, using linkage disequilibrium and haplotypes.
|
Am J Hum Genet
|
2003
|
2.94
|
2
|
Genetically determined P2X7 receptor pore formation regulates variability in chronic pain sensitivity.
|
Nat Med
|
2012
|
2.25
|
3
|
Effect of two- and three-locus linkage disequilibrium on the power to detect marker/phenotype associations.
|
Genetics
|
2004
|
1.85
|
4
|
Multiple tests for genetic effects in association studies.
|
Methods Mol Biol
|
2002
|
1.85
|
5
|
Pain perception is altered by a nucleotide polymorphism in SCN9A.
|
Proc Natl Acad Sci U S A
|
2010
|
1.79
|
6
|
DOMINE: a comprehensive collection of known and predicted domain-domain interactions.
|
Nucleic Acids Res
|
2010
|
1.59
|
7
|
Expansion of the human mu-opioid receptor gene architecture: novel functional variants.
|
Hum Mol Genet
|
2008
|
1.59
|
8
|
Effectiveness of computational methods in haplotype prediction.
|
Hum Genet
|
2001
|
1.53
|
9
|
Potential genetic risk factors for chronic TMD: genetic associations from the OPPERA case control study.
|
J Pain
|
2011
|
1.50
|
10
|
Genetic flip-flop without an accompanying change in linkage disequilibrium.
|
Am J Hum Genet
|
2008
|
1.27
|
11
|
Novel rank-based approaches for discovery and replication in genome-wide association studies.
|
Genetics
|
2011
|
0.99
|
12
|
Large candidate gene association study reveals genetic risk factors and therapeutic targets for fibromyalgia.
|
Arthritis Rheum
|
2012
|
0.97
|
13
|
Haplotype associations with quantitative traits in the presence of complex multilocus and heterogeneous effects.
|
Genet Epidemiol
|
2009
|
0.91
|
14
|
Integrative framework for identification of key cell identity genes uncovers determinants of ES cell identity and homeostasis.
|
Proc Natl Acad Sci U S A
|
2014
|
0.86
|
15
|
Facial pain with localized and widespread manifestations: separate pathways of vulnerability.
|
Pain
|
2013
|
0.86
|