Published in RNA Biol on January 21, 2010
An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype. PLoS Genet (2011) 2.99
Dynamic integration of splicing within gene regulatory pathways. Cell (2013) 2.25
MBNL proteins and their target RNAs, interaction and splicing regulation. Nucleic Acids Res (2014) 1.00
Muscleblind-like 1 (Mbnl1) regulates pre-mRNA alternative splicing during terminal erythropoiesis. Blood (2014) 0.95
Autoregulated splicing of muscleblind-like 1 (MBNL1) Pre-mRNA. J Biol Chem (2011) 0.94
Analysis of exonic regions involved in nuclear localization, splicing activity, and dimerization of Muscleblind-like-1 isoforms. J Biol Chem (2011) 0.94
Reactivation of fetal splicing programs in diabetic hearts is mediated by protein kinase C signaling. J Biol Chem (2013) 0.92
RNA binding proteins in the regulation of heart development. Int J Biochem Cell Biol (2013) 0.84
Muscleblind-like1 undergoes ectopic relocation in the nuclei of skeletal muscles in myotonic dystrophy and sarcopenia. Eur J Histochem (2013) 0.82
Dose-Dependent Regulation of Alternative Splicing by MBNL Proteins Reveals Biomarkers for Myotonic Dystrophy. PLoS Genet (2016) 0.82
Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors. BMC Mol Biol (2012) 0.81
Dissecting domains necessary for activation and repression of splicing by Muscleblind-like protein 1. BMC Mol Biol (2013) 0.81
miR-30-5p Regulates Muscle Differentiation and Alternative Splicing of Muscle-Related Genes by Targeting MBNL. Int J Mol Sci (2016) 0.79
Mechanistic determinants of MBNL activity. Nucleic Acids Res (2016) 0.77
Genome Therapy of Myotonic Dystrophy Type 1 iPS Cells for Development of Autologous Stem Cell Therapy. Mol Ther (2016) 0.76
Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle. PLoS One (2016) 0.76
Muscleblind-like 1 is required for normal heart valve development in vivo. BMC Dev Biol (2015) 0.76
Genome-wide analysis of alternative splicing during human heart development. Sci Rep (2016) 0.75
Autoregulation of MBNL1 function by exon 1 exclusion from MBNL1 transcript. Nucleic Acids Res (2016) 0.75
Derepressing muscleblind expression by miRNA sponges ameliorates myotonic dystrophy-like phenotypes in Drosophila. Sci Rep (2016) 0.75
The importance of CELF control: molecular and biological roles of the CUG-BP, Elav-like family of RNA-binding proteins. Wiley Interdiscip Rev RNA (2011) 1.54
Cloning and embryonic expression patterns of the chicken CELF family. Dev Dyn (2007) 1.01
Muscleblind-like 1 is a negative regulator of TGF-beta-dependent epithelial-mesenchymal transition of atrioventricular canal endocardial cells. Dev Dyn (2009) 0.91
The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation. Nucleic Acids Res (2009) 0.89
Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice. PLoS One (2013) 0.87
Expression of a dominant negative CELF protein in vivo leads to altered muscle organization, fiber size, and subtype. PLoS One (2011) 0.86
CELF-mediated alternative splicing is required for cardiac function during early, but not later, postnatal life. J Mol Cell Cardiol (2008) 0.84
RNA binding proteins in the regulation of heart development. Int J Biochem Cell Biol (2013) 0.84
Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFβ3. BMC Dev Biol (2012) 0.83
Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy. PLoS Curr (2012) 0.82
Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors. BMC Mol Biol (2012) 0.81
Known turnover and translation regulatory RNA-binding proteins interact with the 3' UTR of SECIS-binding protein 2. RNA Biol (2009) 0.80
Differential susceptibility of muscles to myotonia and force impairment in a mouse model of myotonic dystrophy. Muscle Nerve (2011) 0.80