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Ratnadeep Basu
Author PubWeight™ 21.40
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Top papers
Rank
Title
Journal
Year
PubWeight™
‹?›
1
Loss of angiotensin-converting enzyme-2 exacerbates diabetic cardiovascular complications and leads to systolic and vascular dysfunction: a critical role of the angiotensin II/AT1 receptor axis.
Circ Res
2012
1.78
2
Human recombinant ACE2 reduces the progression of diabetic nephropathy.
Diabetes
2009
1.78
3
Angiotensin-converting enzyme 2 suppresses pathological hypertrophy, myocardial fibrosis, and cardiac dysfunction.
Circulation
2010
1.77
4
Loss of p47phox subunit enhances susceptibility to biomechanical stress and heart failure because of dysregulation of cortactin and actin filaments.
Circ Res
2013
1.54
5
Heterozygote loss of ACE2 is sufficient to increase the susceptibility to heart disease.
J Mol Med (Berl)
2014
1.45
6
TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation.
Circ Res
2010
1.32
7
Loss of angiotensin-converting enzyme 2 accelerates maladaptive left ventricular remodeling in response to myocardial infarction.
Circ Heart Fail
2009
1.29
8
Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress.
Circulation
2011
1.12
9
Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.
J Am Heart Assoc
2013
1.05
10
Loss of PI3Kγ enhances cAMP-dependent MMP remodeling of the myocardial N-cadherin adhesion complexes and extracellular matrix in response to early biomechanical stress.
Circ Res
2010
1.03
11
Agonist-induced hypertrophy and diastolic dysfunction are associated with selective reduction in glucose oxidation: a metabolic contribution to heart failure with normal ejection fraction.
Circ Heart Fail
2012
1.02
12
Tissue inhibitor of metalloproteinases (TIMPs) in heart failure.
Heart Fail Rev
2012
1.01
13
Enhanced susceptibility to biomechanical stress in ACE2 null mice is prevented by loss of the p47(phox) NADPH oxidase subunit.
Cardiovasc Res
2011
1.01
14
Cardioprotective effects mediated by angiotensin II type 1 receptor blockade and enhancing angiotensin 1-7 in experimental heart failure in angiotensin-converting enzyme 2-null mice.
Hypertension
2012
0.98
15
Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction.
Am J Physiol Heart Circ Physiol
2010
0.98
16
Angiotensin 1-7 ameliorates diabetic cardiomyopathy and diastolic dysfunction in db/db mice by reducing lipotoxicity and inflammation.
Circ Heart Fail
2014
0.96
17
Pressure-overload-induced heart failure induces a selective reduction in glucose oxidation at physiological afterload.
Cardiovasc Res
2012
0.84
18
Loss of NOX2 (gp91phox) prevents oxidative stress and progression to advanced heart failure.
Clin Sci (Lond)
2014
0.80
19
Reply: RAS Fingerprint.
J Am Coll Cardiol
2017
0.75