Published in Pharmacol Res on January 29, 2010
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Body-wide gene therapy of Duchenne muscular dystrophy in the mdx mouse model. Proc Natl Acad Sci U S A (2006) 1.55
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The mechanism of the force response to stretch in human skinned muscle fibres with different myosin isoforms. J Physiol (2003) 1.19
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A multidisciplinary evaluation of the effectiveness of cyclosporine a in dystrophic mdx mice. Am J Pathol (2005) 1.10
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Multiple pathological events in exercised dystrophic mdx mice are targeted by pentoxifylline: outcome of a large array of in vivo and ex vivo tests. J Appl Physiol (1985) (2009) 1.00
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Investigations of pharmacologic properties of the renal CLC-K1 chloride channel co-expressed with barttin by the use of 2-(p-Chlorophenoxy)propionic acid derivatives and other structurally unrelated chloride channels blockers. J Am Soc Nephrol (2004) 0.99
Fluvastatin and atorvastatin affect calcium homeostasis of rat skeletal muscle fibers in vivo and in vitro by impairing the sarcoplasmic reticulum/mitochondria Ca2+-release system. J Pharmacol Exp Ther (2007) 0.98
Focal adhesion kinase is a load-dependent governor of the slow contractile and oxidative muscle phenotype. J Physiol (2009) 0.98
Major channels involved in neuropsychiatric disorders and therapeutic perspectives. Front Genet (2013) 0.97
Enalapril treatment discloses an early role of angiotensin II in inflammation- and oxidative stress-related muscle damage in dystrophic mdx mice. Pharmacol Res (2011) 0.96
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The KATP channel is a molecular sensor of atrophy in skeletal muscle. J Physiol (2010) 0.93
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Different ability of clenbuterol and salbutamol to block sodium channels predicts their therapeutic use in muscle excitability disorders. Mol Pharmacol (2003) 0.93
Molecular Insights into the Local Anesthetic Receptor within Voltage-Gated Sodium Channels Using Hydroxylated Analogs of Mexiletine. Front Pharmacol (2012) 0.93