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Xiaofa Qin
Author PubWeight™ 21.73
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Top papers
Rank
Title
Journal
Year
PubWeight™
‹?›
1
Gut microbiota: a new aspect that should be taken into more consideration when assessing the toxicity of chemicals or the adverse effects and efficacy of drugs.
Regul Toxicol Pharmacol
2008
1.38
2
Hypoxia-inducible factor plays a gut-injurious role in intestinal ischemia reperfusion injury.
Am J Physiol Gastrointest Liver Physiol
2010
1.11
3
Inactivation of digestive proteases by deconjugated bilirubin: the possible evolutionary driving force for bilirubin or biliverdin predominance in animals.
Gut
2007
1.01
4
Loss of the intestinal mucus layer in the normal rat causes gut injury but not toxic mesenteric lymph nor lung injury.
Shock
2010
0.94
5
Intestinal mucus layer preservation in female rats attenuates gut injury after trauma-hemorrhagic shock.
J Trauma
2010
0.91
6
Primary sclerosing cholangitis and inflammatory bowel disease: where is the link?
Am J Gastroenterol
2007
0.88
7
Intraluminal nonbacterial intestinal components control gut and lung injury after trauma hemorrhagic shock.
Ann Surg
2014
0.86
8
Anticoagulants influence the in vitro activity and composition of shock lymph but not its in vivo activity.
Shock
2011
0.83
9
Is the incidence of inflammatory bowel disease in the developed countries increasing again? Is that surprising?
Inflamm Bowel Dis
2007
0.81
10
What is human inflammatory bowel disease (IBD) more like: Johne's disease in cattle or IBD in dogs and cats?
Inflamm Bowel Dis
2008
0.80
11
What caused the increase of autoimmune and allergic diseases: a decreased or an increased exposure to luminal microbial components?
World J Gastroenterol
2007
0.80
12
Testosterone depletion or blockade in male rats protects against trauma hemorrhagic shock-induced distant organ injury by limiting gut injury and subsequent production of biologically active mesenteric lymph.
J Trauma
2011
0.79
13
What caused the recent worldwide increase of inflammatory bowel disease: should sucralose be added as a suspect?
Inflamm Bowel Dis
2011
0.79
14
With the great complexity unveiling, can we still decipher the interaction between gut flora and the host in inflammatory bowel disease to find out the mechanism and cause? How?
Inflamm Bowel Dis
2008
0.77
15
Inactivation of digestive proteases: another aspect of gut bacteria that should be taken into more consideration.
World J Gastroenterol
2007
0.76
16
The effect of dietary chemicals on gut bacteria and IBD demands further study.
J Crohns Colitis
2011
0.76
17
Synergic effect of bacterial glycosidases and digestive proteases on mucus degradation and the reduced risk of inflammatory bowel disease-like gut damage in both germ-free and poor hygiene conditions.
Inflamm Bowel Dis
2008
0.76
18
What caused the extra high incidence of inflammatory bowel disease in the industrialized countries in the West: lack of some nutrients or increased intake of some harmful agents?
Inflamm Bowel Dis
2009
0.76
19
Inhibition of gut bacteria by dietary chemicals and the hygiene hypothesis.
Ann Allergy Asthma Immunol
2007
0.76
20
Impaired inactivation of digestive proteases: a factor that may have confounded the efficacy of antibiotics aimed at reducing the exposure to luminal bacteria and their components.
Am J Gastroenterol
2008
0.76
21
Inactivation of digestive proteases: another mechanism that probiotics may have conferred a protection.
Am J Gastroenterol
2007
0.76
22
High incidence of inflammatory bowel disease with improved hygiene and failure to get human-like IBD in laboratory animals.
World J Gastroenterol
2007
0.76
23
Reduced production of digestive proteases and the efficacy of enteral and parenteral nutrition on inflammatory bowel disease.
Inflamm Bowel Dis
2008
0.76
24
Has Crohn's disease really occurred anywhere in the digestive tract?
Inflamm Bowel Dis
2008
0.76
25
How to explain recent multiple reports on the decline of inflammatory bowel disease in Canada.
Can J Gastroenterol Hepatol
2014
0.75
26
Comment on: paediatric inflammatory bowel disease in a multiracial Asian country.
Singapore Med J
2013
0.75
27
Increased Digestive Proteases and Decreased β-Glucuronidase in Feces of Rats Treated with Sucralose and Saccharin-Another Critical Evidence That These Dietary Chemicals May Be Important Causative Factors for Inflammatory Bowel Disease.
Inflamm Bowel Dis
2016
0.75