Callie S Kwartler

Author PubWeight™ 10.38^‹?›

Rank	Title	Journal	Year	PubWeight™‹?›
1	TGFB2 mutations cause familial thoracic aortic aneurysms and dissections associated with mild systemic features of Marfan syndrome.	Nat Genet	2012	2.50
2	TGFBR2 mutations alter smooth muscle cell phenotype and predispose to thoracic aortic aneurysms and dissections.	Cardiovasc Res	2010	1.30
3	Genetic variants promoting smooth muscle cell proliferation can result in diffuse and diverse vascular diseases: evidence for a hyperplastic vasculomyopathy.	Genet Med	2010	1.11
4	Rare, nonsynonymous variant in the smooth muscle-specific isoform of myosin heavy chain, MYH11, R247C, alters force generation in the aorta and phenotype of smooth muscle cells.	Circ Res	2012	1.10
5	Aortic remodeling after transverse aortic constriction in mice is attenuated with AT1 receptor blockade.	Arterioscler Thromb Vasc Biol	2013	0.99
6	Smooth muscle hyperplasia due to loss of smooth muscle α-actin is driven by activation of focal adhesion kinase, altered p53 localization and increased levels of platelet-derived growth factor receptor-β.	Hum Mol Genet	2013	0.97
7	Thoracic aortic disease in tuberous sclerosis complex: molecular pathogenesis and potential therapies in Tsc2+/- mice.	Hum Mol Genet	2010	0.96
8	FOXE3 mutations predispose to thoracic aortic aneurysms and dissections.	J Clin Invest	2016	0.78
9	Notch signaling is antagonized by SAO-1, a novel GYF-domain protein that interacts with the E3 ubiquitin ligase SEL-10 in Caenorhabditis elegans.	Genetics	2011	0.76