|
1
|
Neurod1 suppresses hair cell differentiation in ear ganglia and regulates hair cell subtype development in the cochlea.
|
PLoS One
|
2010
|
1.30
|
|
2
|
Conditional deletion of Atoh1 using Pax2-Cre results in viable mice without differentiated cochlear hair cells that have lost most of the organ of Corti.
|
Hear Res
|
2010
|
1.22
|
|
3
|
The molecular basis of making spiral ganglion neurons and connecting them to hair cells of the organ of Corti.
|
Hear Res
|
2011
|
1.18
|
|
4
|
A novel Atoh1 "self-terminating" mouse model reveals the necessity of proper Atoh1 level and duration for hair cell differentiation and viability.
|
PLoS One
|
2012
|
1.14
|
|
5
|
Neurod1 regulates survival and formation of connections in mouse ear and brain.
|
Cell Tissue Res
|
2010
|
1.12
|
|
6
|
Dissecting the molecular basis of organ of Corti development: Where are we now?
|
Hear Res
|
2011
|
0.95
|
|
7
|
Beyond generalized hair cells: molecular cues for hair cell types.
|
Hear Res
|
2012
|
0.94
|
|
8
|
Evolution and development of the tetrapod auditory system: an organ of Corti-centric perspective.
|
Evol Dev
|
2013
|
0.91
|
|
9
|
Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.
|
PLoS One
|
2012
|
0.88
|
|
10
|
Combining lipophilic dye, in situ hybridization, immunohistochemistry, and histology.
|
J Vis Exp
|
2011
|
0.84
|