PubRank

Caterina Motta

Author PubWeight™ 22.25‹?›

Top papers

Rank Title Journal Year PubWeight™‹?›
1 Interleukin-1β causes anxiety by interacting with the endocannabinoid system. J Neurosci 2012 1.55
2 TNF-α-mediated anxiety in a mouse model of multiple sclerosis. Exp Neurol 2012 1.55
3 Loss of striatal cannabinoid CB1 receptor function in attention-deficit / hyperactivity disorder mice with point-mutation of the dopamine transporter. Eur J Neurosci 2011 1.45
4 Fumarates modulate microglia activation through a novel HCAR2 signaling pathway and rescue synaptic dysregulation in inflamed CNS. Acta Neuropathol 2015 1.28
5 Proposed chronic cerebrospinal venous insufficiency criteria do not predict multiple sclerosis risk or severity. Ann Neurol 2011 1.19
6 Interleukin-1β causes synaptic hyperexcitability in multiple sclerosis. Ann Neurol 2012 1.05
7 The (AAT)n repeat of the cannabinoid CB1 receptor gene influences disease progression in relapsing multiple sclerosis. Mult Scler 2010 1.04
8 Preservation of striatal cannabinoid CB1 receptor function correlates with the antianxiety effects of fatty acid amide hydrolase inhibition. Mol Pharmacol 2010 0.98
9 Tumor necrosis factor is elevated in progressive multiple sclerosis and causes excitotoxic neurodegeneration. Mult Scler 2013 0.96
10 Laquinimod prevents inflammation-induced synaptic alterations occurring in experimental autoimmune encephalomyelitis. Mult Scler 2012 0.92
11 Cognitive and cortical plasticity deficits correlate with altered amyloid-β CSF levels in multiple sclerosis. Neuropsychopharmacology 2010 0.90
12 Cannabinoid CB1 receptors regulate neuronal TNF-α effects in experimental autoimmune encephalomyelitis. Brain Behav Immun 2011 0.85
13 Transient receptor potential vanilloid 1 channels modulate the synaptic effects of TNF-α and of IL-1β in experimental autoimmune encephalomyelitis. Neurobiol Dis 2011 0.85
14 Interleukin-1β causes excitotoxic neurodegeneration and multiple sclerosis disease progression by activating the apoptotic protein p53. Mol Neurodegener 2014 0.85
15 Inflammation inhibits GABA transmission in multiple sclerosis. Mult Scler 2012 0.82
16 Synaptic plasticity and PDGF signaling defects underlie clinical progression in multiple sclerosis. J Neurosci 2013 0.82
17 Cerebrospinal fluid detection of interleukin-1β in phase of remission predicts disease progression in multiple sclerosis. J Neuroinflammation 2014 0.81
18 Opposite roles of NMDA receptors in relapsing and primary progressive multiple sclerosis. PLoS One 2013 0.81
19 Growth factors and synaptic plasticity in relapsing-remitting multiple sclerosis. Neuromolecular Med 2014 0.78
20 A genetic variant of the anti-apoptotic protein Akt predicts natalizumab-induced lymphocytosis and post-natalizumab multiple sclerosis reactivation. Mult Scler 2012 0.78
21 Glatiramer acetate protects against inflammatory synaptopathy in experimental autoimmune encephalomyelitis. J Neuroimmune Pharmacol 2013 0.77
22 Cladribine interferes with IL-1β synaptic effects in experimental multiple sclerosis. J Neuroimmunol 2013 0.76
23 Is cerebral glucose metabolism related to blood-brain barrier dysfunction and intrathecal IgG synthesis in Alzheimer disease?: A 18F-FDG PET/CT study. Medicine (Baltimore) 2016 0.75
24 Treatment options to reduce disease activity after natalizumab: paradoxical effects of corticosteroids. CNS Neurosci Ther 2014 0.75