1
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A phosphorus-based dendrimer targets inflammation and osteoclastogenesis in experimental arthritis.
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Sci Transl Med
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2011
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1.69
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2
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TWEAK/Fn14 interactions are instrumental in the pathogenesis of nephritis in the chronic graft-versus-host model of systemic lupus erythematosus.
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J Immunol
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2007
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1.42
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3
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Silencing of microRNA-21 in vivo ameliorates autoimmune splenomegaly in lupus mice.
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EMBO Mol Med
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2011
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1.38
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4
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Deletion of decay-accelerating factor (CD55) exacerbates autoimmune disease development in MRL/lpr mice.
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Am J Pathol
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2002
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1.29
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5
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The role of toll-like receptors in systemic lupus erythematosus.
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Springer Semin Immunopathol
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2006
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1.16
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6
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The Mer receptor tyrosine kinase is expressed on discrete macrophage subpopulations and mainly uses Gas6 as its ligand for uptake of apoptotic cells.
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Clin Immunol
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2009
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1.16
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7
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RS rearrangement frequency as a marker of receptor editing in lupus and type 1 diabetes.
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J Exp Med
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2008
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1.14
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8
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A longitudinal analysis of SLE patients treated with rituximab (anti-CD20): factors associated with B lymphocyte recovery.
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Clin Immunol
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2008
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1.13
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9
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Accelerated atherosclerosis in ApoE deficient lupus mouse models.
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Clin Immunol
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2008
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1.11
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10
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B cell depletion with anti-CD79 mAbs ameliorates autoimmune disease in MRL/lpr mice.
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J Immunol
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2008
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1.11
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11
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Chronic graft-versus-host in Ig knockin transgenic mice abrogates B cell tolerance in anti-double-stranded DNA B cells.
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J Immunol
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2002
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1.05
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12
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T cell-independent spontaneous loss of tolerance by anti-double-stranded DNA B cells in C57BL/6 mice.
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J Immunol
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2008
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1.04
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13
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The Mer receptor tyrosine kinase is required for the loss of B cell tolerance in the chronic graft-versus-host disease model of systemic lupus erythematosus.
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J Immunol
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2008
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1.04
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14
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Secondary heavy chain rearrangement: a mechanism for generating anti-double-stranded DNA B cells.
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J Exp Med
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2003
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1.03
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15
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B cell receptor editing in tolerance and autoimmunity.
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Ann N Y Acad Sci
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2011
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1.01
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16
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Disrupted Mer receptor tyrosine kinase expression leads to enhanced MZ B-cell responses.
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J Autoimmun
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2010
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0.96
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17
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Decay-accelerating factor ameliorates systemic autoimmune disease in MRL/lpr mice via both complement-dependent and -independent mechanisms.
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Am J Pathol
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2007
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0.96
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18
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The role of host CD4 T cells in the pathogenesis of the chronic graft-versus-host model of systemic lupus erythematosus.
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J Immunol
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2005
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0.90
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19
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A protective role of Mer receptor tyrosine kinase in nephrotoxic serum-induced nephritis.
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Clin Immunol
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2010
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0.89
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20
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Modulation of autoimmunity by TLR9 in the chronic graft-vs-host model of systemic lupus erythematosus.
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J Immunol
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2006
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0.84
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21
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The role of antibodies in inflammatory arthritis.
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Immunol Rev
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2010
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0.83
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22
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Selection of individual VH genes occurs at the pro-B to pre-B cell transition.
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J Immunol
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2011
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0.82
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23
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Antibodies in a heavy chain knock-in mouse exhibit characteristics of early heavy chain rearrangement.
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J Immunol
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2009
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0.81
|
24
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In vivo, multimodal imaging of B cell distribution and response to antibody immunotherapy in mice.
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PLoS One
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2010
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0.81
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25
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Intrinsic unresponsiveness of Mertk-/- B cells to chronic graft-versus-host disease is associated with unmodulated CD1d expression.
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J Autoimmun
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2012
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0.80
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26
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KRN/I-Ag7 mouse arthritis is independent of complement C3.
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J Clin Immunol
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2011
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0.79
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27
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B-cell tolerance defects in the B6.Aec1/2 mouse model of Sjögren's syndrome.
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J Clin Immunol
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2012
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0.79
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28
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B cells require "nurturing" by CD4 T cells during development in order to respond in chronic graft-versus-host model of systemic lupus erythematosus.
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Clin Immunol
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2010
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0.79
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29
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Chronic graft-versus-host reaction is associated with a decrease in Ig light chain receptor editing in bone marrow self-reactive B cells.
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Eur J Immunol
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2004
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0.78
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30
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Mature B cells preferentially lose tolerance in the chronic graft-versus-host disease model of systemic lupus erythematosus.
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J Immunol
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2007
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0.78
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31
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Absence of CD59 exacerbates systemic autoimmunity in MRL/lpr mice.
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J Immunol
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2012
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0.76
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32
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Tumor necrosis factor receptor-associated factor 1 influences KRN/I-Ag7 mouse arthritis autoantibody production.
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J Clin Immunol
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2013
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0.75
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33
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Chronic GVH prevents anergy in bone marrow self-reactive B cells: a selective increase in post-endoplasmic reticulum processing and trafficking to the cell surface of autoreactive IgM receptors.
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Int Immunol
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2003
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0.75
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