Ablation of cellular prion protein does not ameliorate abnormal neural network activity or cognitive dysfunction in the J20 line of human amyloid precursor protein transgenic mice.

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Published in J Neurosci on July 20, 2011

Authors

Moustapha Cissé1, Pascal E Sanchez, Daniel H Kim, Kaitlyn Ho, Gui-Qiu Yu, Lennart Mucke

Author Affiliations

1: Gladstone Institute of Neurological Disease and Department of Neurology, University of California, San Francisco, San Francisco, California 94158, USA.

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