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1
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Activation of β-catenin signaling in androgen receptor-negative prostate cancer cells.
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Clin Cancer Res
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2012
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1.68
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2
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The B subunit of the CCAAT box binding transcription factor complex (CBF/NF-Y) is essential for early mouse development and cell proliferation.
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Cancer Res
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2003
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1.63
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3
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Identification of SOX9 interaction sites in the genome of chondrocytes.
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PLoS One
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2010
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1.26
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4
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BMP4 promotes prostate tumor growth in bone through osteogenesis.
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Cancer Res
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2011
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1.23
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5
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Molecular classification of prostate cancer progression: foundation for marker-driven treatment of prostate cancer.
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Cancer Discov
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2013
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1.18
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6
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Modeling a lethal prostate cancer variant with small-cell carcinoma features.
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Clin Cancer Res
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2011
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1.15
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7
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Inhibition of CBF/NF-Y mediated transcription activation arrests cells at G2/M phase and suppresses expression of genes activated at G2/M phase of the cell cycle.
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Nucleic Acids Res
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2006
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1.05
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8
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CBF/NF-Y controls endoplasmic reticulum stress induced transcription through recruitment of both ATF6(N) and TBP.
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J Cell Biochem
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2008
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0.97
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9
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Prioritizing genes associated with prostate cancer development.
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BMC Cancer
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2010
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0.94
|
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10
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Understanding the lethal variant of prostate cancer: power of examining extremes.
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Cancer Discov
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2011
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0.93
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11
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CCAAT binding factor (CBF) binding mediates cell cycle activation of topoisomerase IIalpha. Conventional CBF activation domains are not required.
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J Biol Chem
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2002
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0.93
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12
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Human p32, interacts with B subunit of the CCAAT-binding factor, CBF/NF-Y, and inhibits CBF-mediated transcription activation in vitro.
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Nucleic Acids Res
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2004
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0.89
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13
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Combined Inhibition of IGF-1R/IR and Src family kinases enhances antitumor effects in prostate cancer by decreasing activated survival pathways.
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PLoS One
|
2012
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0.88
|
|
14
|
The dimerization domain of SOX9 is required for transcription activation of a chondrocyte-specific chromatin DNA template.
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Nucleic Acids Res
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2010
|
0.87
|
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15
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Androgen regulation of 5α-reductase isoenzymes in prostate cancer: implications for prostate cancer prevention.
|
PLoS One
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2011
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0.86
|
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16
|
Inactivation of CBF/NF-Y in postnatal liver causes hepatocellular degeneration, lipid deposition, and endoplasmic reticulum stress.
|
Sci Rep
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2011
|
0.86
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