Published in Dose Response on February 10, 2011
Radiation-hormesis phenotypes, the related mechanisms and implications for disease prevention and therapy. J Cell Commun Signal (2014) 0.83
Dynamics between cancer cell subpopulations reveals a model coordinating with both hierarchical and stochastic concepts. PLoS One (2014) 0.81
First generation stochastic gene episilencing (step1) model and applications to in vitro carcinogen exposure. Dose Response (2011) 0.76
Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses. Proc Natl Acad Sci U S A (2003) 6.82
Pathways of DNA double-strand break repair during the mammalian cell cycle. Mol Cell Biol (2003) 6.36
DNA breakage and repair. Adv Genet (1998) 3.38
A double-strand break repair defect in ATM-deficient cells contributes to radiosensitivity. Cancer Res (2004) 2.55
In vivo formation and repair of DNA double-strand breaks after computed tomography examinations. Proc Natl Acad Sci U S A (2005) 2.30
Mechanism of radiation-induced bystander effects: a unifying model. J Pharm Pharmacol (2008) 2.07
Repair of x-ray-induced DNA double-strand breaks in specific Not I restriction fragments in human fibroblasts: joining of correct and incorrect ends. Proc Natl Acad Sci U S A (1995) 1.99
Ionizing radiation-induced, mitochondria-dependent generation of reactive oxygen/nitrogen. Cancer Res (2001) 1.98
Leukocyte DNA damage after multi-detector row CT: a quantitative biomarker of low-level radiation exposure. Radiology (2007) 1.82
Hierarchy of nonhomologous end-joining, single-strand annealing and gene conversion at site-directed DNA double-strand breaks. Nucleic Acids Res (2008) 1.43
Low-dose irradiation of nontransformed cells stimulates the selective removal of precancerous cells via intercellular induction of apoptosis. Cancer Res (2007) 1.36
Cellular responses to DNA double-strand breaks after low-dose gamma-irradiation. Nucleic Acids Res (2009) 1.34
Molecular interaction map of the p53 and Mdm2 logic elements, which control the Off-On switch of p53 in response to DNA damage. Biochem Biophys Res Commun (2005) 1.33
What histone code for DNA repair? Mutat Res (2008) 1.31
DNA double-strand break repair by homologous recombination. Mutat Res (2004) 1.27
Low dose radiation and intercellular induction of apoptosis: potential implications for the control of oncogenesis. Int J Radiat Biol (2007) 1.23
Intercellular communication of cellular stress monitored by gamma-H2AX induction. Carcinogenesis (2009) 1.23
The loss of gammaH2AX signal is a marker of DNA double strand breaks repair only at low levels of DNA damage. Cell Cycle (2006) 1.21
The bystander effect. Health Phys (2003) 1.20
Bystander and delayed effects after fractionated radiation exposure. Radiat Res (2002) 1.17
Higher-order chromatin structure in DSB induction, repair and misrepair. Mutat Res (2010) 1.17
Chromatin structure and DNA damage repair. Epigenetics Chromatin (2008) 1.16
Inducible response required for repair of low-dose radiation damage in human fibroblasts. Proc Natl Acad Sci U S A (2010) 1.16
Strength of the purifying selection against different categories of the point mutations in the coding regions of the human genome. Hum Mol Genet (2006) 1.10
Five repair pathways in one context: chromatin modification during DNA repair. Biochem Cell Biol (2006) 1.10
Whole-body responses to low-level radiation exposure: new concepts in mammalian radiobiology. Exp Hematol (2007) 1.08
Constitutive nitric oxide acting as a possible intercellular signaling molecule in the initiation of radiation-induced DNA double strand breaks in non-irradiated bystander cells. Oncogene (2006) 1.08
Radiation-stimulated epigenetic reprogramming of adaptive-response genes in the lung: an evolutionary gift for mounting adaptive protection against lung cancer. Dose Response (2009) 1.07
No induction of p53 phosphorylation and few focus formation of phosphorylated H2AX suggest efficient repair of DNA damage during chronic low-dose-rate irradiation in human cells. J Radiat Res (2004) 1.07
Participation of DNA-PKcs in DSB repair after exposure to high- and low-LET radiation. Radiat Res (2010) 1.07
Stochastic thresholds: a novel explanation of nonlinear dose-response relationships for stochastic radiobiological effects. Dose Response (2006) 1.06
Repair and misrepair of site-specific DNA double-strand breaks by human cell extracts. Mutat Res (1993) 1.05
Response of heterogeneous ribonuclear proteins (hnRNP) to ionising radiation and their involvement in DNA damage repair. Int J Radiat Biol (2009) 1.05
Joining of correct and incorrect DNA double-strand break ends in normal human and ataxia telangiectasia fibroblasts. Genes Chromosomes Cancer (2000) 1.03
A biological-based model that links genomic instability, bystander effects, and adaptive response. Mutat Res (2004) 0.99
Inter-individual variation in DNA double-strand break repair in human fibroblasts before and after exposure to low doses of ionizing radiation. Mutat Res (2010) 0.97
A dynamic model for the p53 stress response networks under ion radiation. Amino Acids (2006) 0.94
DNA double-strand break repair signalling: the case of RAD51 post-translational regulation. Cell Signal (2002) 0.94
Evidence that natural selection acts on silent mutation. Biosystems (1983) 0.90
Low-dose radiation and genotoxic chemicals can protect against stochastic biological effects. Nonlinearity Biol Toxicol Med (2004) 0.89
Histone H2AX phosphorylation in normal human cells irradiated with focused ultrasoft X rays: evidence for chromatin movement during repair. Radiat Res (2006) 0.86
Effects of ionizing radiation on cell-to-cell communication. Radiat Res (2010) 0.82
Residential radon appears to prevent lung cancer. Dose Response (2011) 0.95
Special issue introduction. Dose Response (2012) 0.88
Stochastic threshold microdose model for cell killing by insoluble metallic nanomaterial particles. Dose Response (2010) 0.77
First generation stochastic gene episilencing (step1) model and applications to in vitro carcinogen exposure. Dose Response (2011) 0.76
Are some neurons hypersensitive to metallic nanoparticles? Dose Response (2010) 0.75
Assessing potential radiological harm to fukushima recovery workers. Dose Response (2011) 0.75