|
1
|
Orange juice neutralizes the proinflammatory effect of a high-fat, high-carbohydrate meal and prevents endotoxin increase and Toll-like receptor expression.
|
Am J Clin Nutr
|
2010
|
1.85
|
|
2
|
Differential effects of cream, glucose, and orange juice on inflammation, endotoxin, and the expression of Toll-like receptor-4 and suppressor of cytokine signaling-3.
|
Diabetes Care
|
2010
|
1.61
|
|
3
|
An antiinflammatory and reactive oxygen species suppressive effects of an extract of Polygonum cuspidatum containing resveratrol.
|
J Clin Endocrinol Metab
|
2010
|
1.48
|
|
4
|
Orange juice or fructose intake does not induce oxidative and inflammatory response.
|
Diabetes Care
|
2007
|
1.46
|
|
5
|
Prolonged reactive oxygen species generation and nuclear factor-kappaB activation after a high-fat, high-carbohydrate meal in the obese.
|
J Clin Endocrinol Metab
|
2007
|
1.38
|
|
6
|
Exenatide exerts a potent antiinflammatory effect.
|
J Clin Endocrinol Metab
|
2011
|
1.37
|
|
7
|
A resveratrol and polyphenol preparation suppresses oxidative and inflammatory stress response to a high-fat, high-carbohydrate meal.
|
J Clin Endocrinol Metab
|
2011
|
1.35
|
|
8
|
Reduction in endotoxemia, oxidative and inflammatory stress, and insulin resistance after Roux-en-Y gastric bypass surgery in patients with morbid obesity and type 2 diabetes mellitus.
|
Surgery
|
2011
|
1.12
|
|
9
|
Suppressive effect of insulin infusion on chemokines and chemokine receptors.
|
Diabetes Care
|
2010
|
1.03
|
|
10
|
Reduction in inflammation and the expression of amyloid precursor protein and other proteins related to Alzheimer's disease following gastric bypass surgery.
|
J Clin Endocrinol Metab
|
2012
|
0.99
|
|
11
|
Hyperandrogenism exerts an anti-inflammatory effect in obese women with polycystic ovary syndrome.
|
Endocrine
|
2012
|
0.91
|
|
12
|
Insulin suppresses the expression of amyloid precursor protein, presenilins, and glycogen synthase kinase-3beta in peripheral blood mononuclear cells.
|
J Clin Endocrinol Metab
|
2011
|
0.88
|
|
13
|
Inflammation in response to glucose ingestion is independent of excess abdominal adiposity in normal-weight women with polycystic ovary syndrome.
|
J Clin Endocrinol Metab
|
2012
|
0.88
|
|
14
|
Hyperglycemia-induced oxidative stress is independent of excess abdominal adiposity in normal-weight women with polycystic ovary syndrome.
|
Hum Reprod
|
2012
|
0.86
|
|
15
|
Insulin infusion suppresses while glucose infusion induces Toll-like receptors and high-mobility group-B1 protein expression in mononuclear cells of type 1 diabetes patients.
|
Am J Physiol Endocrinol Metab
|
2013
|
0.82
|
|
16
|
Glucose-stimulated oxidative stress in mononuclear cells is related to pancreatic β-cell dysfunction in polycystic ovary syndrome.
|
J Clin Endocrinol Metab
|
2013
|
0.81
|
|
17
|
Hyperandrogenism induces a proinflammatory TNFα response to glucose ingestion in a receptor-dependent fashion.
|
J Clin Endocrinol Metab
|
2014
|
0.78
|
|
18
|
The altered mononuclear cell-derived cytokine response to glucose ingestion is not regulated by excess adiposity in polycystic ovary syndrome.
|
J Clin Endocrinol Metab
|
2014
|
0.77
|