Published in Shock on June 01, 2013
Intravenous infusion of mesenteric lymph from severe intraperitoneal infection rats causes lung injury in healthy rats. World J Gastroenterol (2014) 0.88
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Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil priming. Shock (2012) 1.11
Trauma-hemorrhagic shock mesenteric lymph from rat contains a modified form of albumin that is implicated in endothelial cell toxicity. Shock (2005) 1.06
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Changes in the mesenteric lymph proteome induced by hemorrhagic shock. Shock (2010) 0.97
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Adenosine A2A receptor activation reduces lung injury in trauma/hemorrhagic shock. Crit Care Med (2006) 1.63
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Parasympathetic stimulation via the vagus nerve prevents systemic organ dysfunction by abrogating gut injury and lymph toxicity in trauma and hemorrhagic shock. Shock (2013) 1.51
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Small volume albumin administration protects against hemorrhagic shock-induced bone marrow dysfunction. J Trauma (2004) 1.40
Hematopoietic progenitor cells mobilize to the site of injury after trauma and hemorrhagic shock in rats. J Trauma (2007) 1.35
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Trauma hemorrhagic shock-induced lung injury involves a gut-lymph-induced TLR4 pathway in mice. PLoS One (2011) 1.20
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Intravenous injection of trauma-hemorrhagic shock mesenteric lymph causes lung injury that is dependent upon activation of the inducible nitric oxide synthase pathway. Ann Surg (2007) 1.14
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Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil priming. Shock (2012) 1.11
Pancreatic duct ligation abrogates the trauma hemorrhage-induced gut barrier failure and the subsequent production of biologically active intestinal lymph. Shock (2007) 1.10
A study of the biologic activity of trauma-hemorrhagic shock mesenteric lymph over time and the relative role of cytokines. Surgery (2004) 1.10
The mucus layer is critical in protecting against ischemia-reperfusion-mediated gut injury and in the restitution of gut barrier function. Shock (2011) 1.10
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Lithium induces NF-kappa B activation and interleukin-8 production in human intestinal epithelial cells. J Biol Chem (2001) 1.06
Trauma-hemorrhagic shock mesenteric lymph from rat contains a modified form of albumin that is implicated in endothelial cell toxicity. Shock (2005) 1.06
Lipopolysaccharide-induced enterocyte-derived nitric oxide induces intestinal monolayer permeability in an autocrine fashion. Shock (2002) 1.05
Sex hormones modulate distant organ injury in both a trauma/hemorrhagic shock model and a burn model. Surgery (2005) 1.04
Trauma/hemorrhagic shock mesenteric lymph upregulates adhesion molecule expression and IL-6 production in human umbilical vein endothelial cells. Shock (2002) 1.03
β2-Adrenoreceptors of regulatory lymphocytes are essential for vagal neuromodulation of the innate immune system. FASEB J (2011) 1.03
Mesenteric lymph duct ligation prevents shock-induced RBC deformability and shape changes. J Surg Res (2003) 1.03
Appearance of an erythrocyte population with decreased deformability and hemoglobin content following sepsis. Am J Physiol Heart Circ Physiol (2003) 1.03
Cholinergic regulatory lymphocytes re-establish neuromodulation of innate immune responses in sepsis. J Immunol (2011) 1.02
Shock mesenteric lymph-induced rat polymorphonuclear neutrophil activation and endothelial cell injury is mediated by aqueous factors. J Trauma (2002) 1.01
Trauma-hemorrhagic shock-induced up-regulation of endothelial cell adhesion molecules is blunted by mesenteric lymph duct ligation. Crit Care Med (2004) 1.01
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JAK2 inhibition prevents innate immune responses and rescues animals from sepsis. J Mol Med (Berl) (2010) 1.01
Adenosine A2A receptor activation protects CD4+ T lymphocytes against activation-induced cell death. FASEB J (2010) 1.00
Elemental and intravenous total parenteral nutrition diet-induced gut barrier failure is intestinal site specific and can be prevented by feeding nonfermentable fiber. Crit Care Med (2002) 1.00
Mesenteric lymph duct ligation improves survival in a lethal shock model. Shock (2008) 1.00
Trauma-hemorrhagic shock mesenteric lymph induces endothelial apoptosis that involves both caspase-dependent and caspase-independent mechanisms. Ann Surg (2004) 1.00
Adenosine: a potential mediator of immunosuppression in multiple organ failure. Curr Opin Pharmacol (2002) 0.98
The female intestine is more resistant than the male intestine to gut injury and inflammation when subjected to conditions associated with shock states. Am J Physiol Gastrointest Liver Physiol (2004) 0.98
Nitric oxide directly impairs intestinal barrier function. Shock (2002) 0.98
Factors in intestinal lymph after shock increase neutrophil adhesion molecule expression and pulmonary leukosequestration. J Trauma (2003) 0.98
Hemorrhagic shock induces endothelial cell apoptosis, which is mediated by factors contained in mesenteric lymph. Crit Care Med (2004) 0.98
Burn-induced impairment of cardiac contractile function is due to gut-derived factors transported in mesenteric lymph. Shock (2002) 0.97
Hydrophobicity of mucosal surface and its relationship to gut barrier function. Shock (2008) 0.97
Pancreatic duct ligation reduces lung injury following trauma and hemorrhagic shock. Ann Surg (2004) 0.97
Poly(ADP-ribose) polymerase is a regulator of chemokine production: relevance for the pathogenesis of shock and inflammation. Mol Med (2002) 0.96
Albumin peptide: a molecular marker for trauma/hemorrhagic-shock in rat mesenteric lymph. Peptides (2005) 0.96
Inhibition of angiogenesis by the poly(ADP-ribose) polymerase inhibitor PJ-34. Int J Mol Med (2008) 0.95
Trauma-hemorrhage-induced neutrophil priming is prevented by mesenteric lymph duct ligation. Shock (2002) 0.95
Bone marrow failure in male rats following trauma/hemorrhagic shock (T/HS) is mediated by mesenteric lymph and modulated by castration. Shock (2006) 0.95
Red blood cell dysfunction in septic glucose-6-phosphate dehydrogenase-deficient mice. Am J Physiol Heart Circ Physiol (2004) 0.94
CB2 cannabinoid receptors contribute to bacterial invasion and mortality in polymicrobial sepsis. PLoS One (2009) 0.94
Loss of the intestinal mucus layer in the normal rat causes gut injury but not toxic mesenteric lymph nor lung injury. Shock (2010) 0.94
NHE blockade inhibits chemokine production and NF-kappaB activation in immunostimulated endothelial cells. Am J Physiol Cell Physiol (2002) 0.93
Influence of gut microflora on mesenteric lymph cytokine production in rats with hemorrhagic shock. J Trauma (2002) 0.93
Hypertonic saline improves intestinal mucosa barrier function and lung injury after trauma-hemorrhagic shock. Shock (2002) 0.93
Low tidal volume and high positive end-expiratory pressure mechanical ventilation results in increased inflammation and ventilator-associated lung injury in normal lungs. Anesth Analg (2010) 0.92
Gut injury and gut-induced lung injury after trauma hemorrhagic shock is gender and estrus cycle specific in the rat. J Trauma (2003) 0.92
Attenuation of shock-induced acute lung injury by sphingosine kinase inhibition. J Trauma (2004) 0.92
Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathways. Am J Physiol Lung Cell Mol Physiol (2008) 0.92
Intestinal mucus layer preservation in female rats attenuates gut injury after trauma-hemorrhagic shock. J Trauma (2010) 0.91
Mesenteric lymph duct ligation attenuates lung injury and neutrophil activation after intraperitoneal injection of endotoxin in rats. J Trauma (2008) 0.91
Intravenous injection of mesenteric lymph produced during hemorrhagic shock decreases RBC deformability in the rat. J Trauma (2011) 0.90
Na+/H+ exchanger blockade inhibits enterocyte inflammatory response and protects against colitis. Am J Physiol Gastrointest Liver Physiol (2002) 0.90
Oxidative modification of the intestinal mucus layer is a critical but unrecognized component of trauma hemorrhagic shock-induced gut barrier failure. Am J Physiol Gastrointest Liver Physiol (2012) 0.89