Opposing HDAC4 nuclear fluxes due to phosphorylation by β-adrenergic activated protein kinase A or by activity or Epac activated CaMKII in skeletal muscle fibres.

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Published in J Physiol on May 07, 2013

Authors

Yewei Liu1, Martin F Schneider

Author Affiliations

1: Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, MD 21201-1503, USA.

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