A systematic review of cancer GWAS and candidate gene meta-analyses reveals limited overlap but similar effect sizes.

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Published in Eur J Hum Genet on July 24, 2013

Authors

Christine Q Chang1, Ajay Yesupriya2, Jessica L Rowell3, Camilla B Pimentel4, Melinda Clyne4, Marta Gwinn5, Muin J Khoury6, Anja Wulf7, Sheri D Schully1

Author Affiliations

1: Division of Cancer Control and Population Sciences, National Cancer Institute, Bethesda, Maryland, USA.
2: Centers for Disease Control and Prevention, Atlanta, Georgia, USA.
3: Department of Epidemiology, Rollins School of Public Health, Emory University, Atlanta, Georgia, USA.
4: University of Massachusetts Medical School, Graduate School of Biomedical Sciences, Clinical and Population Health Research Program, Worcester, Massachusetts, USA.
5: McKing Consulting Corporation, Atlanta, Georgia, USA.
6: 1] Division of Cancer Control and Population Sciences, National Cancer Institute, Bethesda, Maryland, USA [2] Office of Public Health Genomics, Centers for Disease Control and Prevention, Atlanta, Georgia, USA.
7: Office of Public Health Genomics, Centers for Disease Control and Prevention, Atlanta, Georgia, USA.

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