Improving response inhibition in Parkinson's disease with atomoxetine.

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Published in Biol Psychiatry on February 07, 2014

Authors

Zheng Ye1, Ellemarije Altena1, Cristina Nombela1, Charlotte R Housden2, Helen Maxwell3, Timothy Rittman1, Chelan Huddleston4, Charlotte L Rae4, Ralf Regenthal5, Barbara J Sahakian6, Roger A Barker1, Trevor W Robbins7, James B Rowe8

Author Affiliations

1: Department of Clinical Neurosciences, University of Cambridge, Cambridge, United Kingdom.
2: Department of Clinical Neurosciences, University of Cambridge, Cambridge, United Kingdom; Cambridge Cognition Ltd, University of Cambridge, Cambridge, United Kingdom; Behavioural and Clinical Neuroscience Institute , University of Cambridge, Cambridge, United Kingdom.
3: Department of Experimental Psychology, University of Cambridge, Cambridge, United Kingdom.
4: Medical Research Council Cognition and Brain Sciences Unit, Cambridge, United Kingdom.
5: Division of Clinical Pharmacology, Rudolf-Boehm-Institute of Pharmacology and Toxicology, University of Leipzig, Leipzig, Germany.
6: Behavioural and Clinical Neuroscience Institute, Cambridge, United Kingdom.
7: Department of Experimental Psychology, University of Cambridge, Cambridge, United Kingdom; Behavioural and Clinical Neuroscience Institute, Cambridge, United Kingdom.
8: Department of Clinical Neurosciences, University of Cambridge, Cambridge, United Kingdom; Medical Research Council Cognition and Brain Sciences Unit, Cambridge, United Kingdom; Behavioural and Clinical Neuroscience Institute, Cambridge, United Kingdom. Electronic address: James.Rowe@mrc-cbu.cam.ac.uk.

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