Tight skin 2 mice exhibit a novel time line of events leading to increased extracellular matrix deposition and dermal fibrosis.

PubWeight™: 0.83^‹?›

🔗 View Article (PMID 24820199)

Published in Matrix Biol on May 10, 2014

Authors

Kristen B Long¹, Carol M Artlett¹, Elizabeth P Blankenhorn²

Author Affiliations

1: Department of Microbiology and Immunology, Drexel University College of Medicine, Philadelphia, PA, USA.
2: Department of Microbiology and Immunology, Drexel University College of Medicine, Philadelphia, PA, USA. Electronic address: Eblanken@Drexelmed.edu.

Articles citing this ↘

Tight Skin 2 Mice Exhibit Delayed Wound Healing Caused by Increased Elastic Fibers in Fibrotic Skin. Adv Wound Care (New Rochelle) (2014) 0.99

Recent advances in the genetics of systemic sclerosis: toward biological and clinical significance. Curr Rheumatol Rep (2015) 0.85

The Tsk2/+ mouse fibrotic phenotype is due to a gain-of-function mutation in the PIIINP segment of the Col3a1 gene. J Invest Dermatol (2014) 0.79

Keloids: Animal models and pathologic equivalents to study tissue fibrosis. Matrix Biol (2016) 0.75

Exploring Wound-Healing Genomic Machinery with a Network-Based Approach. Pharmaceuticals (Basel) (2017) 0.75

Articles by these authors ⇆

Animal models of systemic sclerosis: their utility and limitations. Open Access Rheumatol (2014) 0.75