Functional neuroanatomical evidence for the double-deficit hypothesis of developmental dyslexia.

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Published in Neuropsychologia on June 20, 2014

Authors

Elizabeth S Norton1, Jessica M Black2, Leanne M Stanley3, Hiroko Tanaka4, John D E Gabrieli5, Carolyn Sawyer6, Fumiko Hoeft7

Author Affiliations

1: Department of Brain and Cognitive Sciences and McGovern Institute for Brain Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.
2: Graduate School of Social Work, Boston College, Chestnut Hill, MA 02467, USA.
3: Child and Adolescent Psychiatry, Department of Psychiatry, University of California, San Francisco, CA 94143, USA; Pacific Graduate School of Psychology, Palo Alto University, Palo Alto, CA 94304, USA.
4: Child and Adolescent Psychiatry, Department of Psychiatry, University of California, San Francisco, CA 94143, USA; Department of Neuropsychology, Children's Healthcare of Atlanta, and Emory University School of Medicine, Atlanta, GA 30322, USA.
5: Department of Brain and Cognitive Sciences and McGovern Institute for Brain Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Institute for Medical Engineering & Science, Cambridge, MA 02139, USA.
6: Child and Adolescent Psychiatry, Department of Psychiatry, University of California, San Francisco, CA 94143, USA; Department of Pediatrics, Duke University School of Medicine, Durham, NC 27710, USA.
7: Child and Adolescent Psychiatry, Department of Psychiatry, University of California, San Francisco, CA 94143, USA; Haskins Laboratories, New Haven, CT 06511, USA; Department of Neuropsychiatry, Keio University School of Medicine, Tokyo 160, Japan. Electronic address: fumiko.hoeft@ucsf.edu.

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