Cholesterol esters (CE) derived from hepatic sterol O-acyltransferase 2 (SOAT2) are associated with more atherosclerosis than CE from intestinal SOAT2.

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Published in Circ Res on September 19, 2014

Authors

Jun Zhang1, Janet K Sawyer1, Stephanie M Marshall1, Kathryn L Kelley1, Matthew A Davis1, Martha D Wilson1, J Mark Brown1, Lawrence L Rudel2

Author Affiliations

1: From the Section on Molecular Medicine, Department of Internal Medicine, Wake Forest University School of Medicine, Winston-Salem, NC (J.Z., J.K.S., S.M.M., K.L.K., M.A.D., M.D.W., L.L.R.); and Department of Cellular and Molecular Medicine, Cleveland Clinic Lerner Research Institute, OH (S.M.M., J.M.B.).
2: From the Section on Molecular Medicine, Department of Internal Medicine, Wake Forest University School of Medicine, Winston-Salem, NC (J.Z., J.K.S., S.M.M., K.L.K., M.A.D., M.D.W., L.L.R.); and Department of Cellular and Molecular Medicine, Cleveland Clinic Lerner Research Institute, OH (S.M.M., J.M.B.). lrudel@wakehealth.edu.

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