Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury.

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Published in J Neurosci on December 03, 2014

Authors

Angela R Filous1, Amanda Tran1, C James Howell1, Sarah A Busch1, Teresa A Evans1, William B Stallcup2, Shin H Kang3, Dwight E Bergles3, Seong-il Lee4, Joel M Levine4, Jerry Silver5

Author Affiliations

1: Case Western Reserve University, Department of Neurosciences, Cleveland, Ohio 44106.
2: Tumor Microenvironment Program, Cancer Center, Sanford-Burnham Medical Research Institute, La Jolla, California 92037.
3: The Johns Hopkins School of Medicine, The Solomon H. Snyder Department of Neuroscience, Baltimore, Maryland 21205, and.
4: Department of Neurobiology and Behavior, SUNY at Stony Brook, Stony Brook, New York 11794-5230.
5: Case Western Reserve University, Department of Neurosciences, Cleveland, Ohio 44106, jxs10@cwru.edu.

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