BAMBI elimination enhances alternative TGF-β signaling and glomerular dysfunction in diabetic mice.

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Published in Diabetes on January 09, 2015

Authors

Ying Fan1, Xuezhu Li2, Wenzhen Xiao2, Jia Fu2, Ray C Harris3, Maja Lindenmeyer4, Clemens D Cohen4, Nicolas Guillot2, Margaret H Baron5, Niansong Wang6, Kyung Lee2, John C He7, Detlef Schlondorff8, Peter Y Chuang2

Author Affiliations

1: Department of Medicine, Division of Nephrology, Icahn School of Medicine at Mount Sinai, New York, NY Department of Nephrology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
2: Department of Medicine, Division of Nephrology, Icahn School of Medicine at Mount Sinai, New York, NY.
3: Vanderbilt University School of Medicine, Nashville, TN.
4: Division of Nephrology and Institute of Physiology with Center of Integrative Human Physiology, University Hospital and University of Zurich, Zurich, Switzerland.
5: Tisch Cancer Institute and Department of Medicine, Division of Hematology and Medical Oncology, Icahn School of Medicine at Mount Sinai, New York, NY.
6: Department of Nephrology, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, Shanghai, China.
7: Department of Medicine, Division of Nephrology, Icahn School of Medicine at Mount Sinai, New York, NY Renal Section, James J. Peter Veterans Administration Medical Center, Bronx, NY.
8: Department of Medicine, Division of Nephrology, Icahn School of Medicine at Mount Sinai, New York, NY detlef.schlondorff@mssm.edu.

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