β-Glucan exacerbates allergic asthma independent of fungal sensitization and promotes steroid-resistant TH2/TH17 responses.

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Published in J Allergy Clin Immunol on April 20, 2016

Authors

Zhonghua Zhang1, Jocelyn M Biagini Myers1, Eric B Brandt1, Patrick H Ryan2, Mark Lindsey1, Rachael A Mintz-Cole1, Tiina Reponen3, Stephen J Vesper4, Frank Forde1, Brandy Ruff1, Stacey A Bass1, Grace K LeMasters5, David I Bernstein6, James Lockey3, Alison L Budelsky7, Gurjit K Khurana Hershey8

Author Affiliations

1: Division of Asthma Research, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio.
2: Division of Biostatistics and Epidemiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio.
3: Department of Environmental Health, University of Cincinnati College of Medicine, Cincinnati, Ohio.
4: Environmental Protection Agency, Cincinnati, Ohio.
5: Division of Asthma Research, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio; Department of Environmental Health, University of Cincinnati College of Medicine, Cincinnati, Ohio.
6: Department of Internal Medicine, University of Cincinnati College of Medicine, Cincinnati, Ohio.
7: Department of Inflammation Research, Amgen, Seattle, Wash.
8: Division of Asthma Research, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio. Electronic address: Gurjit.Hershey@cchmc.org.

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